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Actinic keratosis (AK), sometimes called solar keratosis or senile keratosis, [1] [2] is a pre-cancerous [3] area of thick, scaly, or crusty skin. [ 4 ] [ 5 ] Actinic keratosis is a disorder ( -osis ) of epidermal keratinocytes that is induced by ultraviolet (UV) light exposure ( actin- ).
Actinic keratoses. What it looks like: Actinic keratoses are pre-cancerous changes to the skin caused by sun damage, explains Dr. Zeichner. “The rash feels like sandpaper when you run your ...
SCC of the skin begins as a small nodule and as it enlarges the center becomes necrotic and sloughs and the nodule turns into an ulcer, and generally are developed from an actinic keratosis. Once keratinocytes begin to grow uncontrollably, they have the potential to become cancerous and produce cutaneous squamous-cell carcinoma. [22]
Grover's disease (GD) is a polymorphic, pruritic, papulovesicular dermatosis characterized histologically by acantholysis [2]: 529 with or without dyskeratosis. [3] Once confirmed, most cases of Grover's disease last six to twelve months, which is why it was originally called "transient".
Lichenoid actinic keratosis; Lichenoid keratosis (benign lichenoid keratosis, lichen planus-like keratosis, solitary lichen planus, solitary lichenoid keratosis) Linear verrucous epidermal nevus (linear epidermal nevus, verrucous epidermal nevus) Malignant acrospiroma (spiradenocarcinoma) Malignant mixed tumor (malignant chondroid syringoma)
In atrophic LP, loss of the rete ridges and dermal fibrosis is prominent. In vesiculobullous LP, the disease progression is quicker. Hence, some of the distinctive features such as hyperkeratosis, hypergranulosis, or dense lymphocytic dermal-epidermal infiltrate may not be present.
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