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Hypokalemia; Other names: Hypokalaemia, hypopotassaemia, hypopotassemia: An ECG in a person with a potassium level of 1.1 meq/L showing the classical changes of ST segment depression, inverted T waves, large U waves, and a slightly prolonged PR interval.
Low potassium is caused by increased excretion of potassium, decreased consumption of potassium rich foods, movement of potassium into the cells, or certain endocrine diseases. [3] Excretion is the most common cause of hypokalemia and can be caused by diuretic use, metabolic acidosis , diabetic ketoacidosis , hyperaldosteronism , and renal ...
Horizontal ST depression in V4, V5, V6 leads during a cardiac stress ECG. Other, non-ischemic, causes include: Side effect of digoxin [4] [3] Hypokalemia [4] [3] Right or left ventricular hypertrophy [3] Intraventricular conduction abnormalities (e.g., right or left bundle branch block, WPW, etc.) [3] Hypothermia [4] Tachycardia [4] Reciprocal ...
Electrocardiography is the process of producing an electrocardiogram (ECG or EKG [a]), a recording of the heart's electrical activity through repeated cardiac cycles. [4] It is an electrogram of the heart which is a graph of voltage versus time of the electrical activity of the heart [ 5 ] using electrodes placed on the skin.
According to V. Gorshkov-Cantacuzene: "The U wave is the momentum carried by the blood in the coronary arteries and blood vessels". [4] [5] [6]The resistivity of stationary blood is expressed as () = | (+), where is a coefficient, and is the hematocrit; at that time, as during acceleration of the blood flow occurs a sharp decrease in the longitudinal resistance with small relaxation times.
EADs can be triggered by hypokalemia and drugs that prolong the QT interval, including class Ia and III antiarrhythmic agents, as well as catecholamines. [1] Afterhyperpolarizations can also occur in cortical pyramidal neurons. There, they typically follow an action potential and are mediated by voltage gated sodium or chloride channels.
Myelosuppression, peripheral neuropathy, anaphylaxis, nausea and vomiting (30-90%), hypokalaemia, metabolic acidosis, interstitial lung disease (uncommon), ototoxicity (rare), reversible posterior leucoencephalopathy syndrome (rare), immune-mediated cytopenias (rare) and hepatic sinusoidal obstruction syndrome (rare). 1.11 Miscellaneous others
Lead II ECG showing a TdP patient being shocked by an implantable cardioverter-defibrillator back to their baseline cardiac rhythm. The following is a partial list of factors associated with an increased tendency towards developing torsades de pointes: [15] Medications; Hypokalemia (low serum potassium) Hypomagnesemia (low serum magnesium)