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The intercostal nerves are part of the somatic nervous system, and arise from the anterior rami of the thoracic spinal nerves from T1 to T11. [1] [2] The intercostal nerves are distributed chiefly to the thoracic pleura and abdominal peritoneum, and differ from the anterior rami of the other spinal nerves in that each pursues an independent course without plexus formation.
The intercostobrachial nerve is the name applied to the lateral cutaneous branch of the second intercostal nerve. It arises anterior to the long thoracic nerve . It provides sensory innervation to the skin of the axilla, and a variable region of the medial side of the upper arm.
The intercostal nerves that give rami to the chest and to the upper parts of the abdominal wall efferent motor innervation and to the pleura and peritoneum afferent sensory innervation are the only ones that do not originate from a plexus. The ventral rami of L1–L5 spinal nerves with a contribution of T12 form lumbar plexus.
It passes through the axilla, at first lying behind, and then medial to the axillary vein, and communicates with the intercostobrachial nerve.. It descends along the medial side of the brachial artery to the middle of the arm, where it pierces the deep fascia, and is distributed to the skin of the back of the lower third of the arm, extending as far as the elbow, where some filaments are lost ...
The anterior divisions of the seventh, eighth, ninth, tenth, and eleventh thoracic intercostal nerves are continued anteriorly from the intercostal spaces into the abdominal wall; hence they are named thoraco-abdominal nerves (or thoracicoabdominal intercostal nerves).
Anterior cutaneous nerve entrapment syndrome (ACNES) is a nerve entrapment condition that causes chronic pain of the abdominal wall. [1] It occurs when nerve endings of the lower thoracic intercostal nerves (7–12) are 'entrapped' in abdominal muscles, causing a severe localized nerve (neuropathic) pain that is usually experienced at the front of the abdomen.
Physicians can also treat intercostal nerve pain by intentionally damaging the intercostal nerves. This process, known as neurolysis , prevents the nerves from sending pain signals. [ 2 ] In chemical neurolysis, a needle injects alcohol or phenol into the nerve and prevents the conduction of pain signals.
The Batson venous plexus, which communicates the posterior intercostal vessels with the vertebral plexus, lacks valves so blood can flow in both directions. The clinical importance of this venous communication is that it represents an important phase in the establishment of vertebral metastases [ 6 ] and neuroschistomiasis.