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268 11705 Ensembl ENSG00000104899 ENSMUSG00000035262 UniProt P03971 P27106 Q5EC55 RefSeq (mRNA) NM_000479 NM_007445 RefSeq (protein) NP_000470 NP_031471 Location (UCSC) Chr 19: 2.25 – 2.25 Mb Chr 10: 80.64 – 80.64 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Anti-Müllerian hormone (AMH), also known as Müllerian-inhibiting hormone (MIH), is a glycoprotein hormone structurally ...
Anti-Müllerian hormone receptor, also known as Müllerian Inhibiting Substance Type II Receptor, is a receptor for the anti-Müllerian hormone (AMH). Furthermore, anti-Müllerian hormone receptor type 2 is a protein in humans that is encoded by the AMHR2 gene .
The sex based differences in the contributions of the paramesonephric ducts to reproductive organs is based on the presence, and degree of presence, of anti-Müllerian hormone. During the formation of the reproductive system, the paramesonephric ducts are formed just lateral to the mesonephric ducts in both female and male embryos 6 weeks after ...
Anti-Müllerian hormone, a protein that affects sexual development of human male embryos; Atlantic Modal Haplotype, a genetic Y-chromosome haplotype; Australian Medicines Handbook, a medical reference text used by health professionals in Australia; Atypical melanocytic hyperplasia
The following is a list of hormones found in Homo sapiens.Spelling is not uniform for many hormones. For example, current North American and international usage uses [citation needed] estrogen and gonadotropin, while British usage retains the Greek digraph in oestrogen and favours the earlier spelling gonadotrophin.
Subsequent development of one set and degeneration of the other depends on the presence or absence of two testicular hormones: testosterone and anti-Müllerian hormone (AMH). Disruption of typical development may result in the development of both, or neither, duct system, which may produce morphologically intersex individuals.
Alfred Jost (1916–1991) was a French endocrinologist, and an early researcher in the field of fetal endocrinology. [1] He is known for his discovery of the Müllerian inhibitor, now called anti-Müllerian hormone (AMH) or Müllerian inhibiting substance (MIS). [2]
The genetic mutational cause of PMDS, is a 27 base-pair deletion of the Anti-Müllerian Type 2 Receptor gene. The 27-base-pair deletion that occurs PMDS is in exon 10 on one allele. [ 9 ] With the AMHR2 gene mutation (PMDS Type 2), the AMHR2 is either not produced, produced in deficient amounts, defective, or the Müllerian ducts manifested a ...