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This type of nerve damage may cause paralysis of the motor, sensory, and autonomic functions, and is mainly seen in crush injury. [2] If the force creating the nerve damage is removed in a timely fashion, the axon may regenerate, leading to recovery. Electrically, the nerve shows rapid and complete degeneration, with loss of voluntary motor units.
The outcome of nerve repair is dependent on the degree of the nerve injury and the circumstances at the site of injury. [5] Since neurapraxia is the least serious form of peripheral nerve injury, recovery and treatment are not extensive. Once the cause of neurapraxia is eliminated, recovery of the lesions in the nerve occurs within a short time ...
The length and efficiency of recovery is depended on the regenerative process that may require 6 to 18 months. The length of the nerve and site of the injury influences the recovery time. To avoid tension during recovery (generally 10–14 days), minimizing movement of the nerve may reduce risk of further damage. [1]
Damage to the myelin sheath of nerves is a nerve injury. It's classified as neuropraxia or a type 1 nerve injury using the Sunderland classification. [2] It can cause a local conduction block for weeks to months as the myelin sheaths regrow, assuming no reinjury which would prolong recovery. [2]
Nerve injury; Fluorescent micrographs (100x) of Wallerian degeneration in cut and crushed peripheral nerves. Left column is proximal to the injury, right is distal. A and B: 37 hours post cut. C and D: 40 hours post crush. E and F: 42 hours post cut. G and H: 44 hours post crush. Specialty: Neurology
Axonotmesis mainly follows a stretch injury. These stretch injuries can either dislocate joints or fracture a limb, due to which peripheral nerves are severed. If the sharp pain from the exposed axon of the nerve is not observed, one can identify a nerve injury from abnormal sensations in their limb.