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Platelets or thrombocytes ... Mitochondrial hyperpolarization is a key event in initiating morphology changes. [25] Intraplatelet calcium concentration increases ...
Giant platelet disorder; Two giant platelets (stained purple) are visible in this image from a light microscope (40×) from a peripheral blood smear surrounded by red blood cells. One normal platelet can be seen in the upper left side of the image (purple) and is significantly smaller in size than the red blood cells (stained pink). Specialty ...
Platelets are held within these internal membranes within the cytoplasm of megakaryocytes. There are two proposed mechanisms for platelet release. In one scenario, these proto-platelet processes break up explosively to become platelets. [7] It is possible to visualize the spontaneous release of platelets using holotomographic live-cell imaging ...
Plant morphology "represents a study of the development, form, and structure of plants, and, by implication, an attempt to interpret these on the basis of similarity of plan and origin". [4] There are four major areas of investigation in plant morphology, and each overlaps with another field of the biological sciences.
Thrombopoietin is a glycoprotein hormone produced by the liver and kidney which regulates the production of platelets. It stimulates the production and differentiation of megakaryocytes, the bone marrow cells that bud off large numbers of platelets. [5] Megakaryocytopoiesis is the cellular development process that leads to platelet production.
Leaf morphology: Shape, margin and venation. Leaf Parts: – A complete leaf is composed of a blade, petiole, and stipules, but in many plants one or more might be lacking or highly modified. Blade – see lamina. Lamina – the flat and laterally-expanded portion of a leaf blade. Leaflet – a separate blade, among others, of a compound leaf
Glanzmann's thrombasthenia is an abnormality of the platelets. [2] It is an extremely rare coagulopathy (bleeding disorder due to a blood abnormality), in which the platelets contain defective or low levels of glycoprotein IIb/IIIa (GpIIb/IIIa), which is a receptor for fibrinogen.
The GM-CSF and IL-3 both work together to stimulate production of all lines. When erythropoietin (EPO) is present, red blood cell production from the CFU-GEMM will be activated. G-CSF, M-CSF, IL-5, IL-4, and IL-3 stimulate the production of neutrophils, monocytes, eosinophils, basophils, and platelets, respectively. [4]