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Mouth breathing as a result of decreased nasal breathing also increases lung surface exposure to irritants, pollutants, and allergens, causing neutrophilic inflammation in response to reactive oxygen species formation; research has found that individuals with genetically hindered glutathione counteraction of this oxidative stress are likely at ...
During pregnancy the plasma volume increases by 40-50% and the red blood cell volume increases only by 20–30%. [22] These changes occur mostly in the second trimester and prior to 32 weeks gestation. [24] Due to dilution, the net result is a decrease in hematocrit or hemoglobin, which are measures of red blood cell concentration.
Shortness of breath (SOB), known as dyspnea (in AmE) or dyspnoea (in BrE), is an uncomfortable feeling of not being able to breathe well enough. The American Thoracic Society defines it as "a subjective experience of breathing discomfort that consists of qualitatively distinct sensations that vary in intensity", and recommends evaluating dyspnea by assessing the intensity of its distinct ...
Prenatal stress can increase the likelihood of maternal and endocrinological problems. Prenatal stress can even cause the embryo to arrive earlier than expected. Sandman and Davis studied "125 full- term infants at 3, 6, and 12 months of age" [ 8 ] to determine the effects of maternal cortisol timing differences on development.
In the case of spina bifida myelomeningocele, the fetus may experience changes to their brain structure among other developmental problems. [7] The second trimester is also essential for perinatal brain development. By week 20 of pregnancy, the area of the brain that is responsible for the awareness of the fetus's five sense begins to develop.
With emphysema the shortness of breath due to effective bronchoconstriction from excessive very thick mucus blockage (it is so thick that great difficulty is encountered in expelling it resulting in near exhaustion at times) can bring on panic attacks unless the individual expects this and has effectively learned pursed lip breathing to more quickly transfer oxygen to the blood via the damaged ...
Exercise intolerance is a condition of inability or decreased ability to perform physical exercise at the normally expected level or duration for people of that age, size, sex, and muscle mass. [1] It also includes experiences of unusually severe post-exercise pain, fatigue, nausea, vomiting or other negative effects.
Respiratory adaptation begins almost immediately after the initiation of the physical stress associated with exercise. This triggers signals from the motor cortex that stimulate the respiratory center of the brain stem, in conjunction with feedback from the proprioreceptors in the muscles and joints of the active limbs.