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  2. GLUT1 - Wikipedia

    en.wikipedia.org/wiki/GLUT1

    Mutations in the GLUT1 gene are responsible for GLUT1 deficiency or De Vivo disease, which is a rare autosomal dominant disorder. [14] This disease is characterized by a low cerebrospinal fluid glucose concentration (hypoglycorrhachia), a type of neuroglycopenia, which results from impaired glucose transport across the blood–brain barrier.

  3. GLUT1 deficiency - Wikipedia

    en.wikipedia.org/wiki/GLUT1_deficiency

    The GLUT1 protein that transports glucose across the blood brain barrier is encoded by the SLC2A1 gene, located on chromosome 1. [8] In GLUT1 deficiency syndrome, one of the two genes is damaged by a mutation and an insufficient amount protein is made. As a result, insufficient glucose is passing the blood brain barrier.

  4. Glucose transporter - Wikipedia

    en.wikipedia.org/wiki/Glucose_transporter

    Levels in cell membranes are increased by reduced glucose levels and decreased by increased glucose levels. GLUT1 expression is upregulated in many tumors. GLUT2: Is a bidirectional transporter, allowing glucose to flow in 2 directions. Is expressed by renal tubular cells, liver cells and pancreatic beta cells.

  5. Vesicular glutamate transporter 1 - Wikipedia

    en.wikipedia.org/wiki/Vesicular_glutamate...

    72961 Ensembl ENSG00000104888 ENSMUSG00000070570 UniProt Q9P2U7 Q3TXX4 RefSeq (mRNA) NM_020309 NM_182993 RefSeq (protein) NP_064705 NP_892038 Location (UCSC) Chr 19: 49.43 – 49.44 Mb Chr 7: 44.81 – 44.83 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Vesicular glutamate transporter 1 (VGLUT1) is a protein that in humans is encoded by the SLC17A7 gene. The protein encoded by this ...

  6. Paroxysmal exercise-induced dystonia - Wikipedia

    en.wikipedia.org/wiki/Paroxysmal_exercise...

    A suspected contributor to familial PED is a mutation in the GLUT1 gene, SLC2A1, which codes for the transporter GLUT1, a protein responsible for glucose entry across the blood–brain barrier. [5] It is not thought that the mutation causes a complete loss of function of the protein but rather only slightly reduces the transporter's activity. [8]

  7. Glucose uptake - Wikipedia

    en.wikipedia.org/wiki/Glucose_uptake

    As a result, GLUT1 facilitates a consistent glucose uptake from the bloodstream, ensuring a steady supply to tissues that rely on glucose. GLUT2 in contrast has a high Km value (15-20mM) and therefore a low affinity for glucose. They are located in the plasma membranes of hepatocytes and pancreatic beta cells (in mice, but GLUT1 in human beta ...

  8. Glutamate dehydrogenase 1 - Wikipedia

    en.wikipedia.org/wiki/Glutamate_dehydrogenase_1

    14661 Ensembl ENSG00000148672 ENSMUSG00000021794 UniProt P00367 P26443 RefSeq (mRNA) NM_005271 NM_001318900 NM_001318901 NM_001318902 NM_001318904 NM_001318905 NM_001318906 NM_008133 RefSeq (protein) NP_001305829 NP_001305830 NP_001305831 NP_001305833 NP_001305834 NP_001305835 NP_005262 NP_032159 Location (UCSC) Chr 10: 87.05 – 87.09 Mb Chr 14: 34.03 – 34.07 Mb PubMed search Wikidata View ...

  9. Glutamate transporter - Wikipedia

    en.wikipedia.org/wiki/Glutamate_transporter

    Glutamate transporters are a family of neurotransmitter transporter proteins that move glutamate – the principal excitatory neurotransmitter – across a membrane.The family of glutamate transporters is composed of two primary subclasses: the excitatory amino acid transporter (EAAT) family and vesicular glutamate transporter (VGLUT) family.