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Increases gastric emptying and small bowel motility Neuropeptide Y: Enteric nerves: Regulation of intestinal blood flow Neurotensin: Ileum: Affects gut motility; increases jejunal and ileal fluid secretion Pancreatic polypeptide: Pancreas: Inhibits pancreatic and biliary secretion Peptide YY: Colon: Inhibits food intake Somatostatin: Stomach ...
5-10% of gastric secretion occurs during this phase. [2] The intestinal phase is a stage in which the duodenum responds to arriving chyme and moderates gastric activity through hormones and nervous reflexes. The duodenum initially enhances gastric secretion, but soon inhibits it.
An enterogastrone is any hormone secreted by the mucosa of the duodenum in the lower gastrointestinal tract in response to dietary lipids that inhibits the caudal (or "forward, analward") motion of the contents of chyme. The function of enterogastrone is almost the same as gastric inhibitor peptide, it inhibits gastric secretion and motility of ...
It can also be stimulated by a pH of 3-4 in the duodenum and by a pH of 1.5 in the stomach. Upon initiation of the reflex, the release of gastrin by G-cells in the antrum of the stomach is shut off. This in turn inhibits gastric motility and the secretion of gastric acid . [1]
The effects range from excitatory or inhibitory effects on motility and secretion to feelings of satiety or hunger when acting on the brain. These hormones fall into three major categories, the gastrin and secretin families, with the third composed of all the other hormones unlike those in the other two families.
CCK mediates digestion in the small intestine by inhibiting gastric emptying. It stimulates the acinar cells of the pancreas to release a juice rich in pancreatic digestive enzymes (hence an alternate name, pancreozymin) that catalyze the digestion of fat, protein, and carbohydrates. Thus, as the levels of the substances that stimulated the ...
glucagon-like peptide-1 (GLP-1) receptor agonists — these stimulate insulin secretion, inhibit the production of glucagon and slow down stomach emptying; they have the added benefit of ...
Also, the secretion of secretin is increased by the products of protein digestion bathing the mucosa of the upper small intestine. [22] Secretin release is inhibited by H 2 antagonists, which reduce gastric acid secretion. As a result, if the pH in the duodenum increases above 4.5, secretin cannot be released. [23]