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Cortical blindness refers to any partial or complete visual deficit that is caused by damage to the visual cortex in the occipital lobe. Unilateral lesions can lead to homonymous hemianopias and scotomas. Bilateral lesions can cause complete cortical blindness and can sometimes be accompanied by a condition called Anton-Babinski syndrome. [26]
Cortical blindness is the total or partial loss of vision in a normal-appearing eye caused by damage to the brain's occipital cortex. [1] Cortical blindness can be acquired or congenital, and may also be transient in certain instances. [ 2 ]
A case of cerebral achromatopsia, acquired after a cortical lesion, was described by Dr. Verrey in 1888. [5] but the evidence was dismissed by both Gordon Holmes and Salomon Henschen, two eminent neurologists who had identified the position and limits of the primary visual cortex. Verrey's description all but disappeared until a color center ...
Hallucinatory palinopsia is a dysfunction of visual memory, caused by localized cortical hyperexcitability or hyperactivity in the posterior visual pathway. Post-geniculate cortical lesions or seizures may cause cortical deafferentation, focal cortical irritation, and epileptic discharges, the proposed mechanisms of hallucinatory palinopsia.
The distribution of the lesions could be linked to the clinical evolution [25] Post-mortem autopsies reveal that gray matter demyelination occurs in the motor cortex, cingulate gyrus, cerebellum, thalamus and spinal cord. [26] Cortical lesions have been observed specially in people with SPMS but they also appear in RRMS and clinically isolated ...
Anton syndrome, also known as Anton-Babinski syndrome and visual anosognosia, is a rare symptom of brain damage occurring in the occipital lobe.Those who have it are cortically blind, but affirm, often quite adamantly and in the face of clear evidence of their blindness, that they are capable of seeing.
Blindsight is the ability of people who are cortically blind to respond to visual stimuli that they do not consciously see due to lesions in the primary visual cortex, also known as the striate cortex or Brodmann Area 17. [1] The term was coined by Lawrence Weiskrantz and his colleagues in a paper published in a 1974 issue of Brain. [2]
The posterior cortical hot zone includes sensory cortical areas in the parietal, temporal, and occipital lobes. It is the “sensory” cortex, much as the frontal cortex is the “action” cortex. When parts of the posterior cortex are damaged, whole modalities of sensory experience disappear from both waking and dreaming .