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Drawing of sclerotic lesions from Babinski's thesis "Etude anatomique et clinique de la sclérose en plaques", 1885. Multiple sclerosis (MS) can be pathologically defined as the presence of distributed glial scars in the central nervous system that must show dissemination in time (DIT) and in space (DIS) to be considered MS lesions.
Pattern II The scar presents T-cells and macrophages around blood vessels, with preservation of oligodendrocytes, as before, but also signs of complement system activation can be found. [60] Though this pattern could be considered similar to damage seen in NMO, some authors report no AQP4 damage in pattern II lesions [61] Pattern III
A vulnerable plaque is a kind of atheromatous plaque – a collection of white blood cells (primarily macrophages) and lipids (including cholesterol) in the wall of an artery – that is particularly unstable and prone to produce sudden major problems such as a heart attack or stroke.
The plaques are highly variable in shape and size; in tissue sections immunostained for Aβ, they comprise a log-normal size distribution curve, with an average plaque area of 400-450 square micrometers (μm 2). The smallest plaques (less than 200 μm 2), which often consist of diffuse deposits of Aβ, [4] are particularly numerous. [6]
Atherosclerosis is characterized by the build-up of plaque inside your blood vessels. It can lead to stenosis, which is a narrowing of your artery walls. Plaque is a fatty substance made up of:
Glucose spikes can also weaken blood vessels, cause plaque in the arteries, ... For people without diabetes, the goal is to gather data, identify patterns, and make changes to your diet, exercise ...