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Very rarely, both lungs may be affected by a pneumothorax. [6] It is often called a "collapsed lung", although that term may also refer to atelectasis. [1] A primary spontaneous pneumothorax is one that occurs without an apparent cause and in the absence of significant lung disease. [3]
In 2022, a major review of tipping points concluded an AMOC collapse would lower global temperatures by around 0.5 °C (0.90 °F) while regional temperatures in Europe would fall by between 4 °C (7.2 °F) and 10 °C (18 °F). [14] [100] A 2020 study assessed the effects of an AMOC collapse on farming and food production in Great Britain. [162]
The lung collapses, impairing normal breathing. Surrounding structures may also shift. When severe enough to cause these shifts and hypotension, it is called a tension pneumothorax. This is life-threatening. The increased pressure inside the chest can compress the heart and lead to a collapse of the blood vessels that drain to the heart.
[2] [3] Changes in the strength of the AMOC are thought to have been responsible for significant changes in past climate. [4] A collapse of the AMOC would have large consequences on the temperatures in the North-Atlantic region. It could lead to a reduction of air temperatures up to 10 °C.
Atelectasis is the partial collapse or closure of a lung resulting in reduced or absence in gas exchange. It is usually unilateral, affecting part or all of one lung. [2] It is a condition where the alveoli are deflated down to little or no volume, as distinct from pulmonary consolidation, in which they are filled with liquid.
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Contusion involves hemorrhage in the alveoli (tiny air-filled sacs responsible for absorbing oxygen), but a hematoma is a discrete clot of blood not interspersed with lung tissue. [4] A collapsed lung can result when the pleural cavity (the space outside the lung) accumulates blood or air (pneumothorax) or both (hemopneumothorax). These ...
The pathophysiology of acute respiratory distress syndrome involves fluid accumulation in the lungs not explained by heart failure (noncardiogenic pulmonary edema). It is typically provoked by an acute injury to the lungs that results in flooding of the lungs' microscopic air sacs responsible for the exchange of gases such as oxygen and carbon dioxide with capillaries in the lungs. [1]