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The Golgi tendon reflex [1] (also called inverse stretch reflex, autogenic inhibition, [2] tendon reflex [3]) is an inhibitory effect on the muscle resulting from the muscle tension stimulating Golgi tendon organs (GTO) of the muscle, and hence it is self-induced.
Reciprocal inhibition is a neuromuscular process in which muscles on one side of a joint relax to allow the contraction of muscles on the opposite side, enabling smooth and coordinated movement. [1] This concept, introduced by Charles Sherrington , a pioneering neuroscientist , is also referred to as reflexive antagonism in some allied health ...
The switch to autogenic excitation is a form of positive feedback. [10] The ascending or afferent pathways to the cerebellum are the dorsal and ventral spinocerebellar tracts. They are involved in the cerebellar regulation of movement. [citation needed]
The transitions between activated and inhibited states can occur via a number of mechanisms. For example, spike-frequency adaptation in the bursting neuron(s) may slowly release the other neuron(s) from inhibition. [15] Reciprocal inhibition is a core feature of many CPGs, including those involved in locomotion. [16] [17] [18] [19]
A common example of reciprocal innervation, is the effect of the nociceptive (or nocifensive) reflex, or defensive response to pain, otherwise commonly known as the withdrawal reflex; a type of involuntary action of the body to remove the body part from the vicinity of an offending object by contracting the appropriate muscles (usually flexor ...
As the muscle tone increases, resistance against flexion of the limb increases as well. However, when flexion is continued, further stretching of the triceps muscle activates an inverse stretch reflex that relaxes the muscle due to autogenic inhibition.
The autogenic inhibition reflex is a spinal reflex phenomenon that involves the Golgi tendon organ. [14] When tension is applied to a muscle, group Ib fibers that innervate the Golgi tendon organ are activated. These afferent fibers project onto the spinal cord and synapse with the spinal interneurons called Ib inhibitory interneurons. [14]
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