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Lymphocyte homing occurs in four steps leading to extravasation into target tissue; Rolling, activation, activation-dependent “arrest”, and diapedesis. [5] Mediated by lymphocyte receptors and vascular ligand interactions, “tethering” is a reversible linkage that leads to either rolling along the vessel wall or transient immediate arrest.
This creates an unstable interaction where the lymphocyte is lightly held to the endothelium wall. The force of the lymph causes the cell to characteristically roll along the vessel. An example of this is the interaction between L-selectin and the PNAD (peripheral node addressin) ligand. Arrest of the 'rolling' cell by activation step.
Neutrophils extravasate from blood vessels to the site of tissue injury or infection during the innate immune response.. In immunology, leukocyte extravasation (also commonly known as leukocyte adhesion cascade or diapedesis – the passage of cells through the intact vessel wall) is the movement of leukocytes (white blood cells) out of the circulatory system (extravasation) and towards the ...
L-selectin, also known as CD62L, is a cell adhesion molecule found on the cell surface of leukocytes, and the blastocyst.It is coded for in the human by the SELL gene. L-selectin belongs to the selectin family of proteins, which recognize sialylated carbohydrate groups containing a Sialyl LewisX (sLeX) determinant. [5]
3576 n/a Ensembl ENSG00000169429 n/a UniProt P10145 n/a RefSeq (mRNA) NM_000584 NM_001354840 n/a RefSeq (protein) NP_000575 NP_001341769 n/a Location (UCSC) Chr 4: 73.74 – 73.74 Mb n/a PubMed search n/a Wikidata View/Edit Human Interleukin 8 (IL-8 or chemokine (C-X-C motif) ligand 8, CXCL8) is a chemokine produced by macrophages and other cell types such as epithelial cells, airway smooth ...
Membrane attack complex (Terminal complement complex C5b-9) A membrane attack complex attached to a pathogenic cell The membrane attack complex (MAC) or terminal complement complex (TCC) is a complex of proteins typically formed on the surface of pathogen cell membranes as a result of the activation of the host's complement system, and as such is an effector of the immune system.