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This category of cysts takes over areas of necrotic tissue in the brain from injuries, diseases, or abnormalities, which occur due to the central nervous system's nonregenerative nature. These cysts can affect all germ layers of the CNS, but are most common in the arachnoid mater , and the ventricular space , which may block CSF pathways.
From their common origin at a joint or tendon, ganglion cysts may form in a wide range of locations. Rarely, intraosseous ganglion cysts occur, sometimes in combination with a cyst in the overlying soft tissue. [6] [16] Rare cases of intramuscular ganglion cysts in the gastrocnemius muscle of the calf have been reported.
Enhancement patterns are highly variable, ranging from minimal to marked, and may be solid, rim, or nodular. Adjacent cord edema and syringomyelia and peritumoral cysts may be present in addition to reactive scoliosis. It is nearly impossible to differentiate ganglioglioma from other more common intramedullary neoplasms based on imaging alone.
Current treatment options include CSF aspiration, fibrin-glue therapy, laminectomy with wrapping of the cyst, among other surgical treatment approaches. Interventional treatment of Tarlov cysts is the only means by which symptoms might permanently be resolved due to the fact that the cysts often refill after aspiration.
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The term "ganglion" refers to the peripheral nervous system. [1] However, in the brain (part of the central nervous system), the basal ganglia are a group of nuclei interconnected with the cerebral cortex, thalamus, and brainstem, associated with a variety of functions: motor control, cognition, emotions, and learning. [citation needed]
Colloid cysts are small fluid-filled sacs located around the middle of the brain. They can become dangerous if left untreated, and treatment is not straightforward.
Treatment Immunomodulators Sensory neuronopathy (also known as sensory ganglionopathy) is a type of peripheral neuropathy that results primarily in sensory symptoms (such as parasthesias , pain or ataxia ) due to destruction of nerve cell bodies in the dorsal root ganglion . [ 1 ]