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Adhesions form as a natural part of the body's healing process after surgery in a similar way that a scar forms. The term "adhesion" is applied when the scar extends from within one tissue across to another, usually across a virtual space such as the peritoneal cavity.
Encapsulating peritoneal sclerosis (EPS) is a chronic clinical syndrome with an insidious onset that manifests as chronic undernourishment accompanied by sporadic, acute, or subacute gastrointestinal obstruction symptoms. [1]
An adhesion barrier is a medical implant that can be used to reduce abnormal internal scarring following surgery by separating the internal tissues and organs while they heal. Surgeons have realized that proper surgical technique is crucial to reduce adhesion formation.
This peritoneal lining of the cavity supports many of the abdominal organs and serves as a conduit for their blood vessels, lymphatic vessels, and nerves. The abdominal cavity (the space bounded by the vertebrae , abdominal muscles , diaphragm , and pelvic floor ) is different from the intraperitoneal space (located within the abdominal cavity ...
Those with bowel resection or lysis of adhesions usually stay in the hospital a few more days until they can eat and walk. [39] Small bowel obstruction caused by Crohn's disease, peritoneal carcinomatosis, sclerosing peritonitis, radiation enteritis, and postpartum bowel obstruction are typically treated conservatively, i.e. without surgery.
The peritoneal cavity is widely used in intraperitoneal injections to administer chemotherapy drugs, [5] [6] and is also utilized in peritoneal dialysis. [7] An increase in capillary pressure in the abdominal organs can cause fluid to leave the interstitial space and enter the peritoneal cavity, resulting in a condition called ascites.
Focal adhesions frequently tether the greater omentum to the cephalad aspect of the transverse mesocolon. The left colon is associated with a similar anatomic configuration of peritoneal folds; the splenic peritoneal fold is contiguous with the left lateral paracolic peritoneal fold at the splenic flexure.
Predisposition is easily aggravated by any of the following: poor motility of the digestive tract, [3] retroperitional tumors, loss of appetite, malabsorption, cachexia, exaggerated lumbar lordosis, visceroptosis, abdominal wall laxity, peritoneal adhesions, abdominal trauma, [10] rapid linear adolescent growth spurt, weight loss, starvation ...