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The respiratory bronchioles run for considerable lengths and become increasingly alveolated with side branches of alveolar ducts that become deeply lined with alveoli. The ducts number between two and eleven from each bronchiole. [10] Each duct opens into five or six alveolar sacs into which clusters of alveoli open.
The respiratory bronchioles deliver air to the exchange surfaces of the lungs. [5] They are interrupted by alveoli which are thin walled evaginations. Alveolar ducts are side branches of the respiratory bronchioles. The respiratory bronchioles are lined by ciliated cuboidal epithelium along with some non-ciliated cells called club cells. [6]
The primary purpose of the respiratory system is the equalizing of the partial pressures of the respiratory gases in the alveolar air with those in the pulmonary capillary blood (Fig. 11). This process occurs by simple diffusion , [ 22 ] across a very thin membrane (known as the blood–air barrier ), which forms the walls of the pulmonary ...
The lungs can be included in the lower respiratory tract or as separate entity and include the respiratory bronchioles, alveolar ducts, alveolar sacs, and alveoli. [3] Adult and pediatric airway anatomy. The respiratory tract can also be divided into a conducting zone and a respiratory zone, based on the distinction of transporting gases or ...
Collateral ventilation is a back-up system of alveolar ventilation that can bypass the normal route of airflow when airways are restricted or obstructed. The pathways involved include those between adjacent alveoli ( pores of Kohn ), between bronchioles and alveoli (canals of Lambert), and those between bronchioles (channels of Martin).
Thus, it includes the alveolar ducts, sacs, and alveoli but not the respiratory bronchioles. [30] The unit described as the secondary pulmonary lobule is the lobule most referred to as the pulmonary lobule or respiratory lobule. [25]: 489 [31] This lobule is a discrete unit that is the smallest component of the lung that can be seen without aid ...
The pathophysiology of acute respiratory distress syndrome involves fluid accumulation in the lungs not explained by heart failure (noncardiogenic pulmonary edema). It is typically provoked by an acute injury to the lungs that results in flooding of the lungs' microscopic air sacs responsible for the exchange of gases such as oxygen and carbon dioxide with capillaries in the lungs. [1]
Hypoventilation exists when the ratio of carbon dioxide production to alveolar ventilation increases above normal values – greater than 45mmHg. If pH is also less than 7.35 this is respiratory acidosis. Hyperventilation exists when the same ratio decreases – less than 35mmHg. If the pH is also greater than 7.45 this is respiratory alkalosis ...