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  2. Apolipoprotein E - Wikipedia

    en.wikipedia.org/wiki/Apolipoprotein_E

    Thus, the genotype most at risk for Alzheimer's disease and at an earlier age is APOE4,4. Using genotype APOE3,3 as a benchmark (with the persons who have this genotype regarded as having a risk level of 1.0) and for white populations only, individuals with genotype APOE4,4 have an odds ratio of 14.9 of developing Alzheimer's disease ...

  3. A subset of Alzheimer's cases may be caused by two ... - AOL

    www.aol.com/news/subset-alzheimers-cases-may...

    APOE4 has long been labeled the biggest genetic risk factor for late-in-life Alzheimer’s, with two copies risker than one. About 2% of the global population is estimated to have inherited a copy ...

  4. People with two copies of a risk gene have genetic form of ...

    www.aol.com/news/people-two-copies-risk-gene...

    CHICAGO (Reuters) -People who carry two copies of the APOE4 gene are virtually guaranteed to develop Alzheimer's and face symptoms at an earlier age, researchers reported on Monday in a study that ...

  5. Heart diseases raise dementia risk: Could a common blood ...

    www.aol.com/heart-diseases-raise-dementia-risk...

    The ApoE4 genetic variant is associated with the ... in the brain long before symptoms appear and a gene involved in heparan sulfate production has been identified as an Alzheimer’s risk factor.

  6. LX1001 - Wikipedia

    en.wikipedia.org/wiki/LX1001

    LX1001 is an experimental gene therapy for Alzheimer's disease associated with mutations to the APOE4 gene. The gene therapy is delivered via adeno-associated virus injected into the spinal canal and is intended to increase the expression of APOE2. [1] [2] [3]

  7. Alzheimer's disease - Wikipedia

    en.wikipedia.org/wiki/Alzheimer's_disease

    A specific isoform of apolipoprotein, APOE4, is a major genetic risk factor for Alzheimer's disease. [15] While apolipoproteins enhance the breakdown of beta amyloid, some isoforms are not very effective at this task (such as APOE4), leading to excess amyloid buildup in the brain. [56]