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Cerebellar cognitive affective syndrome (CCAS), also called Schmahmann's syndrome [1] is a condition that follows from lesions (damage) to the cerebellum of the brain. It refers to a constellation of deficits in the cognitive domains of executive function , spatial cognition , language , and affect resulting from damage to the cerebellum.
[31] [32] In particular, the cerebellar cognitive affective syndrome or Schmahmann's syndrome [33] has been described in adults [34] and children. [35] Estimates based on functional mapping of the cerebellum using functional MRI suggest that more than half of the cerebellar cortex is interconnected with association zones of the cerebral cortex ...
Central pain syndrome; Centurion syndrome; Cerebellar cognitive affective syndrome; Cerebellar stroke syndrome; Cerebellopontine angle syndrome; Cerebral salt-wasting syndrome; Cervicocranial syndrome; Charcot–Marie–Tooth disease; CHARGE syndrome; Charles Bonnet syndrome; Chiari–Frommel syndrome; Chiasmal syndrome; Chilaiditi syndrome
Cerebellar cognitive affective syndrome; Cerebellar stroke syndrome; Corneal-cerebellar syndrome; D. Dandy–Walker malformation; G. Gómez–López-Hernández ...
Gillespie syndrome is a heterogeneous disorder, and can be inherited in either an autosomal dominant or recessive manner. [4] [6] Autosomal dominant inheritance indicates that the defective gene responsible for a disorder is located on an autosome, and only one copy of the gene is sufficient to cause the disorder, when inherited from a parent who has the disorder.
A "cerebellar cognitive affective syndrome" has been described resulting in personality change and how the person shows emotions. [39] Lateral prefrontal cortex – Using our emotions, the lateral prefrontal cortex is responsible for helping us reach our goals by suppressing harmful behaviors or selecting productive ones. [40]
Cerebellar cognitive affective syndrome; Clinical Dementia Rating; Clouding of consciousness; Cognitive impairment; Cognitive vulnerability; Corticobasal degeneration;
Ataxia (from Greek α- [a negative prefix] + -τάξις [order] = "lack of order") is a neurological sign consisting of lack of voluntary coordination of muscle movements that can include gait abnormality, speech changes, and abnormalities in eye movements, that indicates dysfunction of parts of the nervous system that coordinate movement, such as the cerebellum.