Search results
Results From The WOW.Com Content Network
The Environmental Protection Agency has reauthorized the use of "cyanide bomb" chemical traps as a method for killing wild animals. EPA reauthorizes use of controversial 'cyanide bombs' as a ...
Cyanide is quantified by potentiometric titration, a method widely used in gold mining. It can also be determined by titration with silver ion. Some analyses begin with an air-purge of an acidified boiling solution, sweeping the vapors into a basic absorber solution. The cyanide salt absorbed in the basic solution is then analyzed. [47]
This chemical is very flammable, highly toxic, and reactive. If ingested or absorbed through skin, it will cause mild nerve agent symptoms directly as well as blood agent symptoms due to release of HCN. If mixed with water, it gives off poisonous hydrogen cyanide fumes and dimethylamidophosphoric acid.
Sodium dichloroisocyanurate (INN: sodium troclosene, troclosenum natricum or NaDCC or SDIC) is a chemical compound widely used as a cleansing agent and disinfectant. [1] It is a colorless, water-soluble solid, produced as a result of reaction of cyanuric acid with chlorine.
The EPA said Thursday that it also found asbestos in samples of debris that fell in surrounding neighborhoods up to 1.5 miles from the fire. Asbestos can cause several types of cancer, including ...
tert-Butyl isocyanide is an organic compound with the formula Me 3 CNC (Me = methyl, CH 3).It is an isocyanide, commonly called isonitrile or carbylamine, as defined by the functional group C≡N-R. tert-Butyl isocyanide, like most alkyl isocyanides, is a reactive colorless liquid with an extremely unpleasant odor.
Sodium cyanide, like other soluble cyanide salts, is among the most rapidly acting of all known poisons. NaCN is a potent inhibitor of respiration , acting on mitochondrial cytochrome oxidase and hence blocking electron transport.
As such, cyanide poisoning is a form of histotoxic hypoxia, because it interferes with the ability of cells to take or use oxygen via oxidative phosphorylation. [25]: 1475 Specifically, cyanide binds to the heme a3-CuB binuclear center of COX [26] (and thus is a non-competitive inhibitor of it).