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  2. Hyperthyroxinemia - Wikipedia

    en.wikipedia.org/wiki/Hyperthyroxinemia

    Thyroxine or tetraiodothyronine (T4) is produced by the thyroid gland. The term is sometimes used to refer to hyperthyroidism, but hyperthyroidism is a more general term. [2] When the level of thyroxine (T4) in the blood exceeds normal range, it can lead to symptoms such as irritability and unexplained weight loss. [3] Types include:

  3. Thyroid function tests - Wikipedia

    en.wikipedia.org/wiki/Thyroid_function_tests

    Total thyroxine is rarely measured, having been largely superseded by free thyroxine tests. Total thyroxine (Total T 4) is generally elevated in hyperthyroidism and decreased in hypothyroidism. [2] It is usually slightly elevated in pregnancy secondary to increased levels of thyroid binding globulin (TBG). [2]

  4. Thyroid hormones - Wikipedia

    en.wikipedia.org/wiki/Thyroid_hormones

    The thyroid system of the thyroid hormones T 3 and T 4 [1] Thyroid hormones are any hormones produced and released by the thyroid gland, namely triiodothyronine (T 3) and thyroxine (T 4). They are tyrosine-based hormones that are primarily responsible for regulation of metabolism. T 3 and T 4 are partially composed of iodine, derived from food. [2]

  5. Common thyroid drug levothyroxine linked to bone mass loss - AOL

    www.aol.com/common-thyroid-drug-levothyroxine...

    Hypothyroidism is diagnosed by looking at the free thyroxine (T4) levels in people with elevated TSH levels, and comparing the ratio between them. People with high TSH and low T4 get a diagnosis ...

  6. Hyperthyroidism - Wikipedia

    en.wikipedia.org/wiki/Hyperthyroidism

    In overt primary hyperthyroidism, TSH levels are low and T 4 and T 3 levels are high. Subclinical hyperthyroidism is a milder form of hyperthyroidism characterized by low or undetectable serum TSH level, but with a normal serum free thyroxine level. [32]

  7. Thyrotoxic periodic paralysis - Wikipedia

    en.wikipedia.org/wiki/Thyrotoxic_periodic_paralysis

    The abnormality in the channel is thought to lead to shifts of potassium into cells, under conditions of high thyroxine (thyroid hormone) levels, usually with an additional precipitant. Treatment of the low levels of potassium in the blood, followed by correction of the hyperthyroidism, leads to complete resolution of the attacks.