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  2. Guanfacine - Wikipedia

    en.wikipedia.org/wiki/Guanfacine

    Guanfacine does not appear to be effective for improving sleep in children with ADHD and behavioral insomnia. [31] Instead, guanfacine worsened certain sleep parameters, for instance total sleep time, in one clinical trial. [31] [32] Guanfacine has been investigated for treatment of withdrawal for opioids, ethanol, and nicotine. [67]

  3. Management of attention deficit hyperactivity disorder

    en.wikipedia.org/wiki/Management_of_attention...

    Attention deficit hyperactivity disorder management options are evidence-based practices with established treatment efficacy for ADHD.Approaches that have been evaluated in the management of ADHD symptoms include FDA-approved pharmacologic treatment and other pharmaceutical agents, psychological or behavioral approaches, combined pharmacological and behavioral approaches, cognitive training ...

  4. Amy Arnsten - Wikipedia

    en.wikipedia.org/wiki/Amy_Arnsten

    Her research has led to two treatments currently in use: guanfacine for disorders such as ADHD and autism, and prazosin for treating PTSD. [5] Arnsten was featured in a 2019 HBO documentary by Sanjay Gupta and Marc Levin called One Nation Under Stress. [6]

  5. ADHD drugs' impact on brain health, quality of life ... - AOL

    www.aol.com/adhd-drugs-impact-brain-health...

    One in four U.S. adults suspects they may have ADHD and drugs are a first line of treatment for this condition. But what does research say about their impact on the brain and quality of life?

  6. Alpha-adrenergic agonist - Wikipedia

    en.wikipedia.org/wiki/Alpha-adrenergic_agonist

    Guanfacine, [4] (preference for alpha2A-subtype of adrenoceptor) Guanabenz (most selective agonist for alpha2-adrenergic as opposed to imidazoline-I1) Guanoxabenz (metabolite of guanabenz) Xylazine(not for human use), [5] Tizanidine; Methyldopa; Methylnorepinephrine; Norepinephrine [6]

  7. Sympatholytic - Wikipedia

    en.wikipedia.org/wiki/Sympatholytic

    Alpha2-adrenergic receptor agonists, such as clonidine and guanfacine, act at noradrenergic autoreceptors to inhibit the firing of cells in the locus ceruleus, effectively reducing the release of brain norepinephrine. [5]

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