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Contamination with - or excessive exposure to - certain chemicals including: nerve agents, (e.g., sarin, VX, Novichok agents). organophosphorus insecticides (e.g., parathion, aldicarb) nicotine poisoning can be thought of as a subset of cholinergic crisis, as it also involves excessive parasympathetic stimulation. [5]
Tears, from the lacrimal glands, collect in this sac during excessive lacrimation. The fluid then flows through the nasolacrimal duct and into the nasopharynx. This drainage results in what is commonly referred to a runny nose during excessive crying or tear production. Injury or fracture of the lacrimal bone can result in posttraumatic ...
Other symptoms can include photophobia, excessive lacrimation, fever, and reduced corneal sensitivity. The syndrome is classically caused by the spread of an infection into the petrous apex of the temporal bone .
Then, there is a brief silence followed by another cry. Hunger is a main stimulant of the basic cry. An anger cry is much like the basic cry; in this cry, more excess air is forced through the vocal cords, making it a louder, more abrupt cry. This type of cry is characterized by the same temporal sequence as the basic pattern but distinguished ...
An excess of tears, as caused by strong emotion, can cause the nose to run. Quality of vision is affected by the stability of the tear film. [11] Types.
Rhinorrhea is also associated with shedding tears (lacrimation), whether from emotional events or from eye irritation. When excess tears are produced, the liquid drains through the inner corner of the eyelids, through the nasolacrimal duct, and into the nasal cavities. As more tears are shed, more liquid flows into the nasal cavities, both ...
Further signs that may be seen on physical examination are most commonly a diffusely enlarged (usually symmetric), nontender thyroid, lid lag, excessive lacrimation due to Graves' ophthalmopathy, arrhythmias of the heart, such as sinus tachycardia, atrial fibrillation, and premature ventricular contractions, and hypertension. [8] [9]
The clinical signs of high level guanitoxin exposure consists mainly of excessive salivation, lacrimation, chromodacryorrhea (in rats), urinary incontinence, muscular weakness, muscle twitching, convulsion, including opisthotonus, and respiratory distress and/or failure, and death. [4] [5]