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[1] The disease appeared to have a genetic component, and, after hereditary diseases known to affect other horse breeds were investigated, it was concluded that this was a newly identified disease. [1] The cause of disease was tracked down using a genome-wide association study, which implicated a region on horse chromosome 25. [1]
The results of many studies suggest that cows with SCC of less than 200,000 are not likely to be infected with major mastitis pathogens, but cows with SCC above 300,000 are probably infected (Smith, 1996). Herds with bulk tank SCC above 200,000 will have varying degrees of subclinical mastitis present. Data from the National Mastitis Council ...
The California mastitis test (CMT) is a simple cow-side indicator of the somatic cell count of milk. It operates by disrupting the cell membrane of any cells present in the milk sample, allowing the DNA in those cells to react with the test reagent, forming a gel. [1] It provides a useful technique for detecting subclinical cases of mastitis.
A genetic test for WFFS was made commercially available in 2013. Approximately 9–11% of Warmblood horses are carriers, with lower carrier frequencies in Thoroughbreds and Knabstruppers. [2] Horses that are heterozygous for WFFS are phenotypically normal, so genetic testing is necessary to prevent breeding carriers.
Horses heterozygous for the Ile118Lys mutation on the equine EDNRB gene—carriers of lethal white syndrome—usually exhibit a white-spotting pattern called "frame", or "frame overo". [ 5 ] [ 7 ] [ 9 ] [ 13 ] Frame is characterized by jagged, sharply defined, horizontally oriented white patches that run along the horse's neck, shoulder, flank ...
Equine polysaccharide storage myopathy (EPSM, PSSM, EPSSM) is a hereditary glycogen storage disease of horses that causes exertional rhabdomyolysis.It is currently known to affect the following breeds American Quarter Horses, American Paint Horses, Warmbloods, Cobs, Dales Ponies, Thoroughbreds, Arabians, New Forest ponies, and a large number of Heavy horse breeds.
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When a horse is heterozygous for the gene, it is a carrier, but healthy and has no clinical signs of the condition. If two carriers are bred together, however, classic Mendelian genetics indicate a 25% chance of any given mating producing a homozygous foal, hence affected by the disease.