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One extensively studied anterograde amnesiac patient, codenamed H.M., demonstrated that despite his amnesia preventing him from learning new declarative information, procedural memory consolidation was still possible, albeit severely reduced in power. He, along with other patients with anterograde amnesia, were given the same maze to complete ...
Henry Gustav Molaison (February 26, 1926 – December 2, 2008), known widely as H.M., was an American who had a bilateral medial temporal lobectomy to surgically resect the anterior two thirds of his hippocampi, parahippocampal cortices, entorhinal cortices, piriform cortices, and amygdalae in an attempt to cure his epilepsy.
Kent Cochrane was born on August 5, 1951, as the oldest of five children. They grew up in the suburbs of Toronto, Ontario.After attending a community college to study business administration, he obtained a quality control job at a manufacturing plant, which he held until the time of his motorcycle accident.
Anterograde amnesia is one type of memory loss where people have difficulty forming new memories after the amnesia-causing event. Anterograde amnesia is one type of memory loss where people have ...
Patient N.A. (born July 9th, 1938) was an American man who developed anterograde amnesia as a result of a fencing accident. He was a patient studied by Larry Squire, a professor of psychiatry, neuroscience and psychology at the University of California. The cause of his amnesia was found to be a thalamic lesion extending to the hypothalamus.
Another example demonstrated by some patients, such as K.C. and H.M, who have medial temporal damage and anterograde amnesia, still have perceptual priming. Priming was accomplished in many different experiments of amnesia, and it was found that the patients can be primed; they have no conscious recall of the event, but the response is there. [20]
For example, anterograde amnesia, from damage of the medial temporal lobe, is an impairment of declarative memory that affects both episodic and semantic memory operations. [16] Originally, Tulving proposed that episodic and semantic memory were separate systems that competed with each other in retrieval.
Vascular thalamic amnesia occurs when the thalamus is affected by Korsakoff's syndrome or damaged by lacunar infarcts or hemorrhages. [1] Another common cause for damage to the thalamus that may contribute to the development of amnesia is a stroke. [ 2 ]