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Ambient temperature is an example of exogenous vasoconstriction. Cutaneous vasoconstriction will occur because of the body's exposure to the severe cold. Examples of endogenous factors include the autonomic nervous system, circulating hormones, and intrinsic mechanisms inherent to the vasculature itself (also referred to as the myogenic response).
The initial treatment aiming at restoring effective blood pressure in patients that have refractory shock typically starts with introducing norepinephrine and dopamine. [24] Vasopressin comes as the second-line agent. [24] However, high-dose therapy is linked to excessive coronary, splanchnic vasoconstriction, and hypercoagulation. [6]
Various vasoactive agents, such as prostanoids, phosphodiesterase inhibitors, and endothelin antagonists, are approved for the treatment of pulmonary arterial hypertension. The use of vasoactive agents for patients with pulmonary hypertension may cause harm and unnecessary expense to persons with left heart disease or hypoxemic types of lung ...
The hunting reaction or hunting response is a process of alternating vasoconstriction and vasodilation in extremities exposed to cold. The term Lewis reaction is used too, named after Thomas Lewis, who first described the effect in 1930. [1] Vasoconstriction occurs first to reduce heat loss, but also results in strong cooling of the extremities.
Agonists of alpha-2 receptors in the vascular smooth muscle lead to vasoconstriction. However, in clinical practice drugs applied intravenously that are agonists of alpha-2 receptors such as clonidine lead to powerful vasodilation, which causes a decrease in blood pressure by presynaptic activation of the receptors in the sympathetic ganglia.
Generalized vasoconstriction usually results in an increase in systemic blood pressure. Pages in category "Vasoconstrictors" The following 38 pages are in this category, out of 38 total.
The primary application for phentolamine is for the control of hypertensive emergencies, most notably due to pheochromocytoma. [5]It also has usefulness in the treatment of cocaine-induced cardiovascular complications, where one would generally avoid β-blockers (e.g. metoprolol), as they can cause unopposed α-adrenergic mediated coronary vasoconstriction, worsening myocardial ischemia and ...
Treatment is aimed at controlling symptoms and improving the interrupted blood flow to the affected area of the body. [2] Medications include: Antithrombotic medication. These are commonly given because thromboembolism is the major cause of arterial embolism. Examples are: