Search results
Results From The WOW.Com Content Network
Simplified control circuit of human thermoregulation. [8]The core temperature of a human is regulated and stabilized primarily by the hypothalamus, a region of the brain linking the endocrine system to the nervous system, [9] and more specifically by the anterior hypothalamic nucleus and the adjacent preoptic area regions of the hypothalamus.
Heat stroke - Defined by a body temperature of greater than 40 °C (104 °F) due to environmental heat exposure with lack of thermoregulation. Symptoms include dry skin, rapid, strong pulse and dizziness. [7] Heat exhaustion - Can be a precursor of heatstroke; the symptoms include heavy sweating, rapid breathing and a fast, weak pulse.
The human body has two methods of thermogenesis, which produces heat to raise the core body temperature. The first is shivering, which occurs in an unclothed person when the ambient air temperature is under 25 °C (77 °F) [dubious – discuss]. [18] It is limited by the amount of glycogen available in the body. [5]
Hyperthermia affects those who are unable to regulate their body heat, mainly due to environmental conditions. The main risk factor for hyperthermia is the lack of ability to sweat. People who are dehydrated or who are older may not produce the sweat they need to regulate their body temperature. [33]
The human body maintains a core body temperature at around 37 °C or 98.6 °F through mechanisms controlled by the thermoregulatory center within the hypothalamus.When the body is exposed to high ambient temperatures, intense physical exertion, or both, the thermoregulatory center will initiate several processes to dissipate more heat: [9] [11] [12]
Hypoxia is a condition in which the body or a region of the body is deprived of adequate oxygen supply at the tissue level. [1] Hypoxia may be classified as either generalized , affecting the whole body, or local , affecting a region of the body. [ 2 ]
The underlying mechanism involves diffuse injury to cells which form the barrier of the microscopic air sacs of the lungs, surfactant dysfunction, activation of the immune system, and dysfunction of the body's regulation of blood clotting. [5] In effect, ARDS impairs the lungs' ability to exchange oxygen and carbon dioxide. [1]
Heat syncope occurs in a warm environment when blood pressure is lowered as the body dilates (widens) arterioles (small blood vessels) in the skin to radiate heat.This condition occurs within five days of acclimatization to higher temperatures, before the blood volume expands. [3]