Ads
related to: lipodermatosclerosis vs stasis dermatitis
Search results
Results From The WOW.Com Content Network
The origin of lipodermatosclerosis is probably multifactorial, involving tissue hypoxia, leakage of proteins into the interstitium, and leukocyte activation. Studies of patients with lipodermatosclerosis have demonstrated significantly decreased concentrations of cutaneous oxygen associated with decreased capillary density.
Stasis dermatitis is diagnosed clinically by assessing the appearance of red plaques on the lower legs and the inner side of the ankle. Stasis dermatitis can resemble a number of other conditions, such as cellulitis and contact dermatitis, and at times needs the use of a duplex ultrasound to confirm the diagnosis or if clinical diagnosis alone is not sufficient.
It represents late sequelae of lipodermatosclerosis where the skin has lost its nutrient blood flow. Lipodermatosclerosis, [8] an indurated plaque in the medial malleolus. Malignancy, [8] malignant degeneration being a rare but important complication of venous disease since tumors that develop in the setting of an ulcer tend to be more aggressive.
Venous ulcer is defined by the American Venous Forum as "a full-thickness defect of skin, most frequently in the ankle region, that fails to heal spontaneously and is sustained by chronic venous disease, based on venous duplex ultrasound testing."
Lipodermatosclerosis (chronic panniculitis with lipomembranous changes, hypodermitis sclerodermiformis, sclerosing panniculitis, stasis panniculitis) Lipohypertrophy; Localized lipodystrophy; Neutrophilic lobular panniculitis; Nodular vasculitis; Non-progressive late-onset linear hemifacial lipoatrophy
Redness, dryness, and itchiness of areas of skin, termed stasis dermatitis or venous eczema [11] Muscle cramps when making sudden movements, such as standing [11] [12] Abnormal bleeding or healing time for injuries in the affected area; Lipodermatosclerosis or shrinking skin near the ankles
Stasis dermatitis, allergic contact dermatitis, acute irritant contact eczema and infective dermatitis have been documented as possible triggers, but the exact cause and mechanism is not fully understood. [7] Several other types of id reactions exist including erythema nodosum, erythema multiforme, Sweet's syndrome and urticaria. [3]
Allergic/contact dermatitis or atopic dermatitis As above. Eosinophils may be present in the dermis and epidermis (eosinophilic spongiosis). [2] Allergic dermatitis Atopic dermatitis Seborrheic dermatitis Typical findings: [5] Focal, usually mild, spongiosis with overlying scale crust, with a few neutrophils; The crust is often centered on a ...