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Fetal alcohol spectrum disorders encompass a range of physical and neurodevelopmental problems which can result from prenatal alcohol exposure. Diagnosis is based on the signs and symptoms in the person and evidence of alcohol use. [1] These diagnoses of fetal alcohol spectrum disorders are currently recognized: Fetal alcohol syndrome (FAS) [1]
David Weyhe Smith (September 24, 1926 – January 23, 1981) was an American pediatrician and dysmorphologist, best known for his pioneering book Recognizable Patterns of Human Malformation and for describing fetal alcohol syndrome.
However, similar to the discussion about the adverse effect of exposure to alcohol in utero ("fetal alcohol spectrum disorder"), a 2019 study proposed the term "Fetal valproate spectrum disorder" (FVSD) because valproate exposure can lead to a wide range of possible presentations, which can be influenced by various factors (including dosage and ...
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Clarren is a fellow of the American Academy of Pediatrics, the American Association for the Advancement of Science, a past president of the FAS study group for the Research Society on Alcoholism, past president of the West Coast Teratology Society, and a member of the Society for Pediatric Research, New York Academy of Science, the Teratology Society.
Fetal alcohol spectrum disorder is a term describing the range of conditions that result from alcohol exposure during pregnancy. These include physical, behavioral, and neurodevelopment effects as well as learning disabilities. FASD may also refer to: Franklin Area School District, a school system located in Franklin, Pennsylvania
Assess the patient to determine if other signs and symptoms are present: flushed face, hot, dry skin, low output, concentrated urine, anorexia, constipation, diarrhea, or vomiting. Older children may complain of sore throat, headaches, aching, and nausea, as well as, other symptoms. [17] Pulse should be checked at distal and proximal sites.
In the study, 2.5% of 4,000 people born in 1991 and 1992 were found by ultrasound scanning at the age of 18 to have non-alcoholic fatty liver disease; five years later transient elastography (fibroscan) found over 20% to have the fatty deposits on the liver of steatosis, indicating non-alcoholic fatty liver disease; half of those were classified as severe.