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Atrial fibrillation is associated with an increased risk of heart failure, dementia, and stroke. [3] [12] It is a type of supraventricular tachycardia. [14] Atrial fibrillation frequently results from bursts of tachycardia that originate in muscle bundles extending from the atrium to the pulmonary veins. [15]
The left atrial appendage is a pouch-like structure located in the upper part of the left atrium. [1] Left atrial appendage occlusion (LAAO) is an alternative therapy to oral anticoagulation in a certain subset of patients with atrial fibrillation. Atrial fibrillation is characterized by an irregular and uncoordinated pumping function of the atria.
The heart has four chambers, two upper chambers—the right and left atrium, and two lower chambers—the right and left ventricles. Fibrillation describes when the muscle fibers are all contracting at different times, so the end result is this quivering, or twitching movement.
High in the upper part of the left atrium is a muscular ear-shaped pouch – the left atrial appendage (LAA) (lat: auricula atrii sinistra), which has a tubular trabeculated structure. [9] LAA anatomy as seen in a CT scan is characterized as being in one of four groups: chicken wing (48%), cactus (30%), windsock (19%), and cauliflower(3%).
[6] [7] GP has been shown to be a contributor to atrial fibrillation (AFib), such that ablation of the GP has been a strategy for treatment of AFib. [1] GP ablation alone has been shown to eliminate AFib in approximately three-quarter of AFib patients. [1] Ligation of the left atrial appendage may reduce AFib by alteration of the GP. [8]
Bachmann's bundle receives its blood supply from the sinoatrial nodal artery (right, left or both). [4]Besides Bachmann's bundle, the other three conduction tracts that constitute the atrial conduction system are known as the anterior, middle, and posterior tracts, which run from the sinoatrial node to the atrioventricular node, converging in the region near the coronary sinus.
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