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Retinal hemorrhage (UK English: retinal haemorrhage) is a disorder of the eye in which bleeding occurs in the retina, the light sensitive tissue located on the back wall of the eye. [1] There are photoreceptor cells in the retina called rods and cones , which transduce light energy into nerve signals that can be processed by the brain to form ...
A tear in the retina can allow fluids from the eye to leak in behind the retina, which causes retinal detachment. When this occurs, blood from the retinal blood vessels can bleed into the vitreous. [4] Retinal tear accounts for 11.4–44% of vitreous hemorrhage cases. [1]
Valsalva retinopathy is a form of sub-retinal, sub-hyaloid or sub-internal limiting membrane hemorrhage occur due to rupture of retinal vessels caused by a strenuous physical activity. [1]
A subconjunctival hemorrhage can often occur without any obvious cause or harm to the eye. A strong enough sneeze or cough can cause a blood vessel in the eye to burst. Hyphema is a result of blunt or penetrating trauma to the orbit that increases intraocular pressure, causing tears in the vessels of the ciliary body and iris.
The risk of retinal detachment is the greatest in the first 6 weeks following a vitreous detachment, but can occur over 3 months after the event.. The risk of retinal tears and detachment associated with vitreous detachment is higher in patients with myopic retinal degeneration, lattice degeneration, and a familial or personal history of previous retinal tears/detachment.
Both types cause disease by altering the normal blood flow to the retina through different mechanisms. The retina is supplied by small vessel branches from the central retinal artery. [8] Proliferative retinopathy refers to damage caused by abnormal blood vessel growth. [9] Normally, angiogenesis is a natural part of tissue growth and formation.
In general, branch retinal vein occlusion has a good prognosis: after 1 year 50–60% of eyes have been reported to have a final visual acuity of 20/40 or better even without any treatment. With time the dramatic picture of an acute branch retinal vein occlusion becomes more subtle, hemorrhages fade so that the retina can look almost normal.
Recently, central serous chorioretinopathy has been understood to be part of the pachychoroid spectrum. [5] [6] In pachychoroid spectrum disorders, of which CSR represents stage II, the choroid, the highly vascularized layer below the retina, is thickened and congested with increased blood vessel diameter, especially in the deep choroid (the so-called Haller's layer).