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  2. Anti-amyloid drugs - Wikipedia

    en.wikipedia.org/wiki/Anti-amyloid_drugs

    Aducanumab, sold under the brand name Aduhelm, is an anti-amyloid drug designed to treat Alzheimer's disease. It is a monoclonal antibody that targets aggregated forms (plaque) of amyloid beta (Aβ) found in the brains of people with Alzheimer's disease to reduce its buildup. [10] It was developed by Biogen and Eisai. [11] Aducanumab is given via intravenous infusion. [5] Aducanumab was ...

  3. Amyloid plaques - Wikipedia

    en.wikipedia.org/wiki/Amyloid_plaques

    Amyloid plaques naturally occur in the aging brains of nonhuman species ranging from birds to great apes. [4] In nonhuman primates, which are the closest biological relatives of humans, plaques have been found in all species examined thus far. [ 47 ]

  4. Dr. Sanjay Gupta is optimizing his brain health. Here are his ...

    www.aol.com/lifestyle/dr-sanjay-gupta-optimizing...

    5. Talk to your doctor about supplements that may boost brain health. Isaacson’s testing showed that the balance of Gupta’s levels of two fatty acids — omega-3 and omega-6 — were out of whack.

  5. Dopamine treatment destroys beta-amyloid plaques in ... - AOL

    www.aol.com/lifestyle/dopamine-treatment...

    “Beta-amyloid plaques are a hallmark of the disease and are thought to contribute to its progression. If L-DOPA can be shown to be effective in reducing beta-amyloid plaques in humans, it could ...

  6. Aducanumab - Wikipedia

    en.wikipedia.org/wiki/Aducanumab

    Aducanumab, sold under the brand name Aduhelm, is an anti-amyloid drug designed to treat Alzheimer's disease. [2] [3] It is a monoclonal antibody [3] [2] that targets aggregated forms (plaque) [4] [5] of amyloid beta (Aβ) found in the brains of people with Alzheimer's disease to reduce its buildup. [6] [7] It was developed by Biogen and Eisai. [8]

  7. LECT2 amyloidosis - Wikipedia

    en.wikipedia.org/wiki/Lect2_amyloidosis

    LECT2 Amyloidosis (ALECT2) is a form of amyloidosis caused by the LECT2 protein. It was found to be the third most common (~3% of total) cause of amyloidosis in a set of more than 4,000 individuals studied at the Mayo Clinic; the first and second most common forms the disorder were AL amyloidosis and AA amyloidosis, respectively.

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