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There are several options of treatment when iatrogenic (i.e., caused by the surgeon) spinal accessory nerve damage is noted during surgery. For example, during a functional neck dissection that injures the spinal accessory nerve, injury prompts the surgeon to cautiously preserve branches of C2, C3, and C4 spinal nerves that provide supplemental innervation to the trapezius muscle. [3]
Injury to the accessory nerve can result in neck pain and weakness of the trapezius muscle. Symptoms will depend on at what point along its length the nerve was severed. [5] Injury to the nerve can result in shoulder girdle depression, atrophy, abnormal movement, a protruding scapula, and weakened abduction. [5]
A major function: controls muscles for voice and resonance and the soft palate. Symptoms of damage: dysphagia (swallowing problems), velopharyngeal insufficiency. This nerve is involved (together with nerve IX) in the pharyngeal reflex or gag reflex. XI Accessory. Sometimes: cranial accessory, spinal accessory. Mainly motor Cranial and Spinal Roots
The accessory deep peroneal nerve is an anomalous nerve in which the nerve splits off from the common peroneal nerve and sometimes innervates the extensor digitorum brevis muscle. By itself, the condition is harmless but in conjunction with other neurological and structural defects in the area, can make the condition more difficult. [ 1 ]
In contrast, pseudobulbar palsy is a clinical syndrome similar to bulbar palsy but in which the damage is located in upper motor neurons of the corticobulbar tracts in the mid-pons (i.e., in the cranial nerves IX-XII), that is the nerve cells coming down from the cerebral cortex innervating the motor nuclei in the medulla.
Denervation may be the result of nerve injury. The three main types of nerve injury are neurapraxia, axonotmesis and neurotmesis. These three types distinguish between the severity of the nerve damage and the potential for recovery after the damage. After an injury in which some nerves are damaged, the brain has shown capabilities in rewiring ...
Muscle wasting, fasciculations and fibrillations are typically signs of end-stage muscle denervation and are seen over a longer time period. Another feature is the segmentation of symptoms – only muscles innervated by the damaged nerves will be symptomatic.
A superior laryngeal nerve palsy changes the pitch of the voice and causes an inability to make explosive sounds due to paralysis of the cricothyroid muscle. If no recovery is evident three months after the palsy initially presents, the damage is most likely to be permanent. A bilateral palsy presents as a tiring and hoarse voice.