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It refers to a characteristic boomerang-shaped area of restricted diffusion in the splenium of the corpus callosum due to cytotoxic edema. [1] This sign is associated with various neurological conditions and is considered a non-specific marker of splenial pathology, often reversible depending on the underlying cause. [2] [3]
Agenesis of the corpus callosum (ACC) is a rare congenital disorder that is one of the most common brain malformations observed in human beings, [30] in which the corpus callosum is partially or completely absent. ACC is usually diagnosed within the first two years of life, and may manifest as a severe syndrome in infancy or childhood, as a ...
Dejerine in 1892 described specific symptoms resulting from a lesion to the corpus callosum that caused alexia without agraphia. The patient had a lesion in the left occipital lobe, blocking sight in the right visual field , and in the splenium of the corpus callosum. Dejerine interpreted this case as a disconnection of the speech area in the ...
The indusium griseum is prolonged around the splenium of the corpus callosum as a delicate layer, the fasciolar gyrus, [3] which is continuous below with the surface of the dentate gyrus. [4] The indusium griseum and fasciolar gyrus are very small components of the limbic lobe , and are continuations of the hippocampal formation, forming an ...
It is secondary association cortex, making connections with numerous other brain regions. The region's name refers to its anatomical location immediately behind the splenium of the corpus callosum in primates, although in rodents it is located more
Split-brain or callosal syndrome is a type of disconnection syndrome when the corpus callosum connecting the two hemispheres of the brain is severed to some degree. It is an association of symptoms produced by disruption of, or interference with, the connection between the hemispheres of the brain.
Pure alexia almost always involves an infarct to the left posterior cerebral artery (which perfuses the splenium of the corpus callosum and left visual cortex, among other things). The resulting deficit will be pure alexia – i.e., the patient can write but cannot read (even what they have just written).
The quadrigeminal cistern lies between the splenium of the corpus callosum (superiorly), the cerebellar vermis (inferiorly and posteriorly), [5] [6] and the tentorial margin. [6] It is just superior to the tectum of the mesencephalon (midbrain). [3] It lies medial to part of the medial occipital cortex. [6]