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  2. Cyclooxygenase-2 inhibitor - Wikipedia

    en.wikipedia.org/wiki/Cyclooxygenase-2_inhibitor

    In the CLASS trial which compared Celebrex 800 mg/day to ibuprofen 2400 mg/day and diclofenac 150 mg/day for osteoarthritis or rheumatoid arthritis for six months, Celebrex was associated with significantly fewer upper gastrointestinal complications (0.44% vs. 1.27%, p = 0.04), with no significant difference in incidence of cardiovascular ...

  3. Celecoxib - Wikipedia

    en.wikipedia.org/wiki/Celecoxib

    Celecoxib, sold under the brand name Celebrex among others, is a COX-2 inhibitor and nonsteroidal anti-inflammatory drug (NSAID). [7] It is used to treat the pain and inflammation in osteoarthritis , acute pain in adults, rheumatoid arthritis , psoriatic arthritis , ankylosing spondylitis , painful menstruation , and juvenile rheumatoid ...

  4. FDA approves new type of non-opioid pain medication, 1st of ...

    www.aol.com/fda-approves-type-non-opioid...

    This is the first class of non-opioid pain medication approved to treat moderate to severe acute pain approved by the FDA in more than 20 years. MORE: Opioids are no better than a placebo for back ...

  5. Celebrex not always better than cheap acetaminophen - AOL

    www.aol.com/2008/02/01/celebrex-not-always...

    When it comes to pain, I'm convinced that the right medication is the one that you believe will work-- anything from the latest designer drug to eye of newt. Fortunately, there are scientists ...

  6. Ibuprofen, by comparison, works faster than naproxen. However, because ibuprofen’s effects wear off quicker, it needs to be taken more frequently throughout the day, she says.

  7. Prostaglandin inhibitors - Wikipedia

    en.wikipedia.org/wiki/Prostaglandin_inhibitors

    Prostaglandin inhibitors are drugs that inhibit the synthesis of prostaglandin in human body. [1] There are various types of prostaglandins responsible for different physiological reactions such as maintaining the blood flow in stomach and kidney, regulating the contraction of involuntary muscles and blood vessels, and act as a mediator of inflammation and pain.

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