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Nucleotide excision repair is a DNA repair mechanism. [2] DNA damage occurs constantly because of chemicals (e.g. intercalating agents ), radiation and other mutagens . Three excision repair pathways exist to repair single stranded DNA damage: Nucleotide excision repair (NER), base excision repair (BER), and DNA mismatch repair (MMR).
Setting a value for any of the cell or organelle attributes will make its diagram visible Any number and combination of diagram attributes may be set When multiple diagrams are activated, the title is suppressed
It is called "non-homologous" because the break ends are directly ligated without the need for a homologous template, in contrast to homology directed repair (HDR), which requires a homologous sequence to guide repair. NHEJ is active in both non-dividing and proliferating cells, while HDR is not readily accessible in non-dividing cells. [1]
Add the new template to the table in the common documentation afterwards. Please consider reusing one of the other templates and please choose the color sensibly. If you find a table cell template that does not take a parameter and you want to be able to change the text in the cell, do not duplicate the template! Instead, edit the template and ...
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The system can also inhibit cell division and cell respiration. [1] The SOS response has been proposed as a model for bacterial evolution of certain types of antibiotic resistance. [2] The SOS response is a global response to DNA damage in which the cell cycle is arrested and DNA repair and mutagenesis are induced.
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