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Traumatic brain injury may cause damage to the hypothalamus or the pituitary gland, and deficiencies of pituitary hormones (hypopituitarism) can cause similar symptoms to post-concussion syndrome; in these cases, symptoms can be treated by replacing any hormones that are deficient. [medical citation needed]
Symptoms of a mild brain injury include headaches, confusions, tinnitus, fatigue, changes in sleep patterns, mood or behavior. Other symptoms include trouble with memory, concentration, attention or thinking. [3] Mental fatigue is a common debilitating experience and may not be linked by the patient to the original (minor) incident.
Prognosis, or the likely progress of a disorder, depends on the nature, location, and cause of the brain damage (see Traumatic brain injury, Focal and diffuse brain injury, Primary and secondary brain injury). In children with uncomplicated minor head injuries the risk of intracranial bleeding over the next year is rare at 2 cases per 1 million ...
The relative risk of post-traumatic seizures (PTS) increases with the severity of traumatic brain injury (TBI). [128] A CT of the head years after a traumatic brain injury showing an empty space where the damage occurred marked by the arrow. Improvement of neurological function usually occurs for two or more years after the trauma.
Post-traumatic amnesia is generally due to a head injury. Traumatic amnesia is often transient, but may be permanent or either anterograde, retrograde, or mixed type. The extent of the period covered by the amnesia is related to the degree of injury and may give an indication of the prognosis for recovery of other functions.
The relative risk of post-traumatic seizures increases with the severity of traumatic brain injury. [20] Pain, especially headache, is a common complication following a TBI. [1] Being unconscious and lying still for long periods can cause blood clots to form (deep venous thrombosis), which can cause pulmonary embolism. [21]
Immunoreactive axonal profiles are observed as either granular (B, G, H) or more elongated, fusiform (F) swellings in the corpus callosum and the brain stem (H) at 24h post traumatic brain injury. Example of APP immunoreactive neurons (arrow heads) observed in the cortex underneath the impact site (E, G). No APP staining was observed in healthy ...
Post-traumatic amnesia (PTA) is a state of confusion that occurs immediately following a traumatic brain injury (TBI) in which the injured person is disoriented and unable to remember events that occur after the injury. [1] The person may be unable to state their name, where they are, and what time it is. [1]