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Coxa valga is a deformity of the hip where the angle formed between the head and neck of the femur and its shaft is increased, usually above 135 degrees.. The deformity may develop in children with neuromuscular disorders (i.e. cerebral palsy, spinal dysraphism, poliomyelitis), skeletal dysplasias, and juvenile idiopathic arthritis.
Coxa vara is a deformity of the hip, whereby the angle between the head and the shaft of the femur is reduced to less than 120 degrees. This results in the leg being shortened and the development of a limp. It may be congenital and is commonly caused by injury, such as a fracture.
Two forms of femoral dysplasia are coxa vara, in which the femur head grows at too narrow an angle to the shaft, and coxa valga, in which the angle is too wide. A rare type, the "Beukes familial hip dysplasia" is found among Afrikaners that are members of the Beukes family. The femur head is flat and irregular.
In vertebrate anatomy, the hip, or coxa [1] (pl.: coxae) in medical terminology, refers to either an anatomical region or a joint on the outer (lateral) side of the pelvis.. The hip region is located lateral and anterior to the gluteal region, inferior to the iliac crest, and lateral to the obturator foramen, with muscle tendons and soft tissues overlying the greater trochanter of the femur. [2]
The human abdomen is divided into quadrants and regions by anatomists and physicians for the purposes of study, diagnosis, and treatment. [1] [2] The division into four quadrants allows the localisation of pain and tenderness, scars, lumps, and other items of interest, narrowing in on which organs and tissues may be involved. The quadrants are ...
Children until the age of 3 to 4 have a degree of genu varum. The child sits with the soles of the feet facing one another; the tibia and femur are curved outwards; and, if the limbs are extended, although the ankles are in contact, there is a distinct space between the knee-joints. During the first year of life, a gradual change takes place.
[1] and is a syndrome of camptodactyly, arthropathy, coxa vara, and pericarditis. [2] It may also include congenital cataracts. [3] The cause of this syndrome was discovered in 1999. [4] Children with this syndrome often present with a joint effusion that is cool and resistant to anti-inflammatory therapy.
Medication is not the primary treatment for hypermobility, but can be used as an adjunct treatment for related joint pain. Nonsteroidal anti-inflammatory drugs are the primary medications of choice. Narcotics are not recommended for primary or long-term treatment and are reserved for short-term use after acute injury.