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Beta blockers are known primarily for their reductive effect on heart rate, although this is not the only mechanism of action of importance in congestive heart failure. [17] Beta blockers, in addition to their sympatholytic β 1 activity in the heart, influence the renin–angiotensin system at the kidneys.
A lower resting heart rate or slower heartbeat will fill the ventricles/heart better and allow for more of a forceful contraction of blood out to the rest of the body, says Dr. Weinberg.
Additionally, the heart rate and contractile force increase when SNS is activated, which may be harmful to cardiac function as it increases metabolic demand. [1] Adrenergic blocking agents treat certain diseases through blocking the adrenergic receptor, [2] [3] preventing it from being activated by noradrenaline and epinephrine. As a result, it ...
This is in contrast to other commonly used rate-reducing medications, such as beta-blockers and calcium channel blockers, which not only reduce heart rate, but also the cardiac contractility. Given the selective decrease in rate without loss of contractility, ivabradine may prove efficacious for treatment of congestive heart failure with ...
Beta-blockers act as competitive antagonists that block the effects of catecholamines at beta-adrenergic receptor sites, resulting in reduced rate and force of contraction of the heart, as well as reduced peripheral vascular resistance. [14] Non-selective beta-blockers: propranolol, nadolol, timolol
Beta-blockers, which slow down the heart and reduce the force of its contractions. Ace Inhibitors, which block the formation of Angiotensin II, a chemical that causes blood vessels to constrict.