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Cortical deafness is a rare form of sensorineural hearing loss caused by damage to the primary auditory cortex.Cortical deafness is an auditory disorder where the patient is unable to hear sounds but has no apparent damage to the structures of the ear (see auditory system).
In one case study, each of the three sound types (music, environmental sounds, speech) was also shown to recover independently (Mendez and Geehan, 1988-case 2 [22]). It is yet unclear whether general auditory agnosia is a combination of milder auditory disorders, or whether the source of this disorder is at an earlier auditory processing stage.
Auditory processing disorder (APD), rarely known as King-Kopetzky syndrome, is a neurodevelopmental disorder affecting the way the brain processes sounds. [2] Individuals with APD usually have normal structure and function of the ear, but cannot process the information they hear in the same way as others do, which leads to difficulties in recognizing and interpreting sounds, especially the ...
Central auditory processing disorder This is not an actual hearing loss but gives rise to significant difficulties in hearing. One kind of auditory processing disorder is King-Kopetzky syndrome , which is characterized by an inability to process out background noise in noisy environments despite normal performance on traditional hearing tests.
Auditory verbal agnosia can also occur as a result of traumatic brain injury. In one case, a man fell and developed auditory verbal agnosia. This can be challenging to diagnose because he had fluent speech and could understand written language. However, he was unable to follow oral commands or repeat words.
Charcot–Marie–Tooth disease [3] an inherited neurological disorder with delayed onset that can affect the ears as well as other organs. The hearing loss in this condition is often ANSD (auditory neuropathy spectrum disorder) a neural cause of hearing loss. Muckle–Wells syndrome, a rare inherited autoinflammatory disorder, can lead to ...
Transcortical sensory aphasia is characterized as a fluent aphasia. Fluency is determined by direct qualitative observation of the patient’s speech to determine the length of spoken phrases, and is usually characterized by a normal or rapid rate; normal phrase length, rhythm, melody, and articulatory agility; and normal or paragrammatic speech. [5]
Based on clinical testing of subjects with auditory neuropathy, the disruption in the stream of sound information has been localized to one or more of three probable locations: the inner hair cells of the cochlea, the synapse between the inner hair cells and the auditory nerve, or a lesion of the ascending auditory nerve itself.