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Mapping of several bone diseases onto levels of vitamin D (calcidiol) in the blood [6] Normal bone vs. osteoporosis. Vitamin D deficiency is typically diagnosed by measuring the concentration of the 25-hydroxyvitamin D in the blood, which is the most accurate measure of stores of vitamin D in the body.
If due to a lack of vitamin D, treatment is usually with vitamin D and calcium. [2] This generally results in improvements within a few weeks. [2] Bone deformities may also improve over time. [5] Occasionally surgery may be performed to correct bone deformities. [8] [3] Genetic forms of the disease typically require specialized treatment. [5]
Vitamin D (the inactive version) is mainly from two forms: vitamin D 3 and vitamin D 2. Vitamin D 3, or cholecalciferol, is formed in the skin after exposure to sunlight or ultra violet radiation or from D 3 supplements or fortified food sources. Vitamin D 2, or ergocalciferol, is obtained from D 2 supplements or fortified food sources. [3]
Vitamin D deficiency, especially in early life, is linked to an increased risk of autoimmune conditions, such as type 1 diabetes. Autoimmune diseases can be caused by a failure of T cells, a type ...
Healthcare professionals can diagnose vitamin D deficiency with a 25-hydroxy vitamin D blood test. This test checks for 25-hydroxyvitamin D, a common form of vitamin D that circulates in the blood.
This condition can be caused by vitamin D, calcium or phosphorus deficiency. [86] Vitamin D deficiency remains the main cause of rickets among young infants in most countries because breast milk is low in vitamin D, and darker skin, social customs, and climatic conditions can contribute to inadequate sun exposure.