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A 12 lead ECG showing sinus tachycardia. Sinus tachycardia is usually apparent on an ECG, but if the heart rate is above 140 bpm the P wave may be difficult to distinguish from the previous T wave and one may confuse it with a paroxysmal supraventricular tachycardia or atrial flutter with a 2:1 block.
A sinus rhythm is any cardiac rhythm in which depolarisation of the cardiac muscle begins at the sinus node. [1] It is necessary, but not sufficient, for normal electrical activity within the heart. [2] On the electrocardiogram (ECG), a sinus rhythm is characterised by the presence of P waves that are normal in morphology. [2]
Sinus tachycardia is another regular rhythm however the ventricular rate is quicker, between 100 - 160 bpm, with a normal PR interval and normal QRS complex. Sinus arrhythmia is an irregular rhythm with a ventricular rate of 60 - 100 normally, however a slow rhythm can be distinguished when the rate is less than 60, the PR interval and QRS ...
Physiologic J-junctional depression with sinus tachycardia [3] Hyperventilation [3] Horizontal ST depression in V4, V5, V6 leads during a cardiac stress ECG. Other, non-ischemic, causes include: Side effect of digoxin [4] [3] Hypokalemia [4] [3] Right or left ventricular hypertrophy [3]
This sinus rhythm is important because it ensures that the heart's atria reliably contract before the ventricles, ensuring as optimal stroke volume and cardiac output. [ 4 ] In junctional rhythm, however, the sinoatrial node does not control the heart's rhythm – this can happen in the case of a block in conduction somewhere along the pathway ...
Inappropriate sinus tachycardia (IST) is a diagnosis of exclusion, [16] a rare but benign type of cardiac arrhythmia that may be caused by a structural abnormality in the sinus node. It can occur in seemingly healthy individuals with no history of cardiovascular disease.
When an individual is in normal sinus rhythm, the ECG characteristics of WPW are a short PR interval (less than 120 milliseconds in duration), widened QRS complex (greater than 120 milliseconds in duration) with slurred upstroke of the QRS complex, and secondary repolarization changes (reflected in ST segment-T wave changes). [citation needed]
ECG would be abnormal in 75 to 95% of the patients. Characteristic ECG changes would be large QRS complex associated with giant T wave inversion [4] in lateral leads I, aVL, V5, and V6, together with ST segment depression in left ventricular thickening. For right ventricular thickening, T waves are inverted from V2 to V3 leads.