Ad
related to: neurogenic vs vascular claudication- 262 Neil Avenue # 430, Columbus, Ohio · Directions · (614) 221-7464
- Find a Doctor
Meet with our experts to diagnose
your symptoms and receive treatment
- Should I see a Heart Doc
Talk to your doc about your heart
and learn what to ask
- Find a Doctor
Search results
Results From The WOW.Com Content Network
Both neurogenic claudication and vascular claudication manifest as leg pain with walking, but several key features help distinguish between these conditions. [7] In contrast to NC, vascular claudication does not vary with changes in posture. [9] Patients with vascular claudication may experience relief with standing, which may provoke symptoms ...
Spinal or neurogenic claudication may be differentiated from arterial claudication based on activity and position. In neurogenic claudication, positional changes lead to increased stenosis (narrowing) of the spinal canal and compression of nerve roots and resultant lower extremity symptoms. Standing and extension of the spine narrows the spinal ...
Intermittent claudication, also known as vascular claudication, is a symptom that describes muscle pain on mild exertion (ache, cramp, numbness or sense of fatigue), [1] classically in the calf muscle, which occurs during exercise, such as walking, and is relieved by a short period of rest.
Vascular claudication can resemble spinal stenosis, and some individuals experience unilateral or bilateral symptoms radiating down the legs rather than true claudication. [7] The first symptoms of stenosis include bouts of low back pain. After a few months or years, this may progress to claudication.
Intermittent neurogenic claudication [17] [20] [21] characterized by lower limb numbness, weakness, diffuse or radicular leg pain associated with paresthesia (bilaterally), [20] weakness and/or heaviness in buttocks radiating into lower extremities with walking or prolonged standing. [17]
Neurogenic TOS includes disorders produced by compression of components of the brachial plexus nerves. The neurogenic form of TOS accounts for 95% of all cases of TOS. [21] Arterial TOS is due to compression of the subclavian artery. [21] This is less than one percent of cases. [2] Venous TOS is due to compression of the subclavian vein. [21]
There are less common differential diagnostic conditions included myelopathy, myopathy, vascular or neurogenic claudication, hypotensive akathisia, orthostatic tremor, painful legs, and moving toes. [12]
Magnesium deficiency causes neurogenic inflammation in a rat model. Researchers have theorized that since substance P which appears at day five of induced magnesium deficiency, is known to stimulate in turn the production of other inflammatory cytokines including IL-1, Interleukin 6 (IL-6), and TNF-alpha (TNFα), which begin a sharp rise at day 12, substance P is a key in the path from ...