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This is why after a stroke people have a chance of developing cognitive deficits that result in anterograde amnesia, since strokes can involve the temporal lobe in the temporal cortex, and the temporal cortex houses the hippocampus. Anterograde amnesia can be the first clinical sign that Alzheimer's disease is developing within the brain ...
Postoperative cognitive dysfunction (POCD) is a decline in cognitive function (especially in memory and executive functions) that may last from 1–12 months after surgery, or longer. [1] In some cases, this disorder may persist for several years after major surgery. [2] POCD is distinct from emergence delirium. Its causes are under ...
Since stroke is an essential part of vascular dementia, [13] the goal is to prevent new strokes. This is attempted through reduction of stroke risk factors, such as high blood pressure, high blood lipid levels, atrial fibrillation, or diabetes mellitus. [2] [5] Medications for high blood pressure are used to prevent pre-stroke dementia. [19]
EF exhibited memory loss of his personal experiences (e.g., childhood), and the impaired ability to recognize his wife and parents. [47] JG is the first recorded patient with isolated RA. [10] GH, a mother and a wife, had surgery in August 2002. When GH woke up after the surgery, she believed it was May 1989. [47]
The most prominent symptom of post-traumatic amnesia (PTA) is a loss of memory of the present time. [10] As a result, patients are often unaware of their condition and may behave as if they are going about their regular lives. This can cause complications if patients are confined to a hospital and may lead to agitation, distress and anxiety. [10]
Amnesia is a deficit in memory caused by brain damage or brain diseases, [1] but it can also be temporarily caused by the use of various sedative and hypnotic drugs. The memory can be either wholly or partially lost due to the extent of damage that is caused.
The memory loss associated with damage to the vascular thalamus tends to maintain high variability from patient to patient. Some patients will maintain their memory while developing the behavioral disorders while others will show signs of declarative anterograde amnesia with no signs of behavioral disorders. [4]
The attack was witnessed by a capable observer and reported as being a definite loss of recent memory (anterograde amnesia). There was an absence of clouding of consciousness or other cognitive impairment other than amnesia. There were no focal neurological signs or deficits during or after the attack.