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  2. Angiotensin - Wikipedia

    en.wikipedia.org/wiki/Angiotensin

    Angiotensin is a peptide hormone that causes vasoconstriction and an increase in blood pressure. It is part of the renin–angiotensin system, which regulates blood pressure. Angiotensin also stimulates the release of aldosterone from the adrenal cortex to promote sodium retention by the kidneys. An oligopeptide, angiotensin is a hormone and a ...

  3. Angiotensin-converting enzyme 2 - Wikipedia

    en.wikipedia.org/.../Angiotensin-converting_enzyme_2

    Membrane bound angiotensin-converting enzyme 2 (mACE2) is a zinc-containing metalloenzyme located on the surface of intestinal enterocytes, renal tubular cells and other cells. [ 6 ] [ 17 ] mACE2 protein contains an N-terminal peptidase M2 domain and a C-terminal collectrin renal amino acid transporter domain.

  4. Angiotensin-converting enzyme - Wikipedia

    en.wikipedia.org/wiki/Angiotensin-converting_enzyme

    Angiotensin-converting enzyme (EC 3.4.15.1), or ACE, is a central component of the renin–angiotensin system (RAS), which controls blood pressure by regulating the volume of fluids in the body. It converts the hormone angiotensin I to the active vasoconstrictor angiotensin II. Therefore, ACE indirectly increases blood pressure by causing blood ...

  5. Renin–angiotensin system - Wikipedia

    en.wikipedia.org/wiki/Renin–angiotensin_system

    The renin-angiotensin system (RAS), or renin-angiotensin-aldosterone system (RAAS), is a hormone system that regulates blood pressure, fluid, and electrolyte balance, and systemic vascular resistance.

  6. Discovery and development of angiotensin receptor blockers

    en.wikipedia.org/wiki/Discovery_and_development...

    ARBs are blocking the last part of the renin–angiotensin pathway and block the pathway more specifically than ACE inhibitors. [1] The AT 1 receptor mediates Ang II to cause increased cardiac contractility, sodium reabsorption and vasoconstriction which all lead to increased blood pressure. By blocking AT 1 receptors, ARBs lead to lower blood ...

  7. Thromboxane A2 - Wikipedia

    en.wikipedia.org/wiki/Thromboxane_A2

    Contrastly, TXA 2 vascular tissue synthesis is stimulated by angiotensin II which promotes cyclooxygenase I's metabolism of arachidonic acid. An angiotensin II dependent pathway also induces hypertension and interacts with TXA 2 receptors. [6]

  8. Angiotensin II receptor - Wikipedia

    en.wikipedia.org/wiki/Angiotensin_II_receptor

    AT 2 receptors are more plentiful in the fetus and neonate. The AT 2 receptor remains enigmatic and controversial – is probably involved in vascular growth. Effects mediated by the AT 2 receptor are suggested to include inhibition of cell growth, fetal tissue development, modulation of extracellular matrix, neuronal regeneration, apoptosis, cellular differentiation, and maybe vasodilation ...

  9. Renal physiology - Wikipedia

    en.wikipedia.org/wiki/Renal_physiology

    It stimulates erythropoiesis (production of red blood cells) in the bone marrow. Calcitriol, the activated form of vitamin D, promotes intestinal absorption of calcium and the renal reabsorption of phosphate. Renin is an enzyme which regulates angiotensin and aldosterone levels.