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In most cases, the wilt phase occurs on seedlings, but for certain corn types (i.e. sweet corn), more mature plants can wilt. The wilt phase is systemic, meaning the majority of the plant is infected via bacterial movement and colonization of the plant's vascular system. When the bacterium spreads within the plant, leaves begin withering and ...
S. kunkelii, the corn stunt spiroplasma, was characterized as the causative agent of corn stunt disease by Whitcomb et al. in 1986. [19] [20] In the literature, the combination of maize bushy stunt mycoplasma and maize rayado fino marafivirus in addition to S. kunkelii has been called the corn stunt disease complex, also called "achaparramiento."
Plants affected later in their reproductive cycle may experience a slowing in ear development (“Maize Dwarf Mosaic Virus,” n.d.), while some plants may even become barren (Lipps & Mills, n.d.). It is not uncommon for plants to have shortened upper internodes or an increase in tiller number (“Maize Dwarf Mosaic Virus,” n.d.).
The absence of a gene found only in plants with Texas male sterile cytoplasm is the reason for this resistance. [1] Corn plants with T-cms cytoplasm have maternally inherited the gene T-urf 13, which encodes for a protein component of the inner mitochondrial membrane. T-toxin acts on this portion of the mitochondria. [4]
Corn grey leaf spot can be an extremely devastating disease as potential yield losses range from 5 to 40 US bushels per acre (440 to 3,480 L/ha). At higher disease levels, even greater losses can result. When a corn plant's ability to store and produce carbohydrates (glucose) in the grain is diminished, yield losses take place.
Spiroplasma kunkelii is also referred to as Corn Stunt Spiroplasma as it is the causative agent of Corn stunt disease, a disease of corn and other grasses that stunts plant growth. Spiroplasma kunkelii represents a major economic risk, as corn production in the United States is an industry worth over $50 billion. [ 25 ]