Ad
related to: megaloblastic anemia other names and pictures of death syndrome people
Search results
Results From The WOW.Com Content Network
Megaloblastic anemia is a type of macrocytic anemia. An anemia is a red blood cell defect that can lead to an undersupply of oxygen. [1] Megaloblastic anemia results from inhibition of DNA synthesis during red blood cell production. [2] When DNA synthesis is impaired, the cell cycle cannot progress from the G2 growth stage to the mitosis (M ...
Imerslund–Gräsbeck syndrome is a rare autosomal recessive, familial form of vitamin B12 deficiency caused by malfunction of the "Cubam" receptor located in the terminal ileum. This receptor is composed of two proteins, amnionless (AMN), and cubilin. A defect in either of these protein components can cause this syndrome.
Macrocytic anemia: Megaloblastic anemia: D51.1, D52.0, D53.1: 29507: Megaloblastic anemia (or megaloblastic anaemia) is an anemia of macrocytic classification that results from inhibition of DNA synthesis in red blood cell production. [10] Microangiopathic hemolytic anemia: Minkowski-Chauffard syndrome: Myelophthisic anemia: D61.9
Macrocytic anemias have several causes but with the implementation of folic acid fortification in North America, folate deficiency has become a rare cause of megaloblastic macrocytic anemia in that part of the world. [1] In this region, Vitamin B 12 deficiency is a far more common cause of megaloblastic macrocytic anemia. [1]
Lubani Al Saleh Teebi syndrome also known as cystic fibrosis-gastritis-megaloblastic anemia syndrome is a very rare autosomal recessive genetic disorder which consists of cystic fibrosis, helicobacter pylori-associated gastritis, folate-deficiency megaloblastic anemia and intellectual disabilities.
The presence of hypersegmented neutrophils is an important diagnostic feature of megaloblastic anaemias. Hypersegmentation can also be seen in many other conditions but with relatively less diagnostic significance. Hypersegmentation can sometimes be difficult to assert since interobserver variation is high and segmentation may vary with race.
Pernicious anemia as described in 1849 by Addison is now also known as Addison-Biermer disease. It is a type of megaloblastic anemia, in which a lack of intrinsic factor causes absorption of vitamin B 12 to be impaired. It is caused by a lack of parietal cells in the stomach. [13]
Immune mediated hemolytic anemia (direct Coombs test is positive) Autoimmune hemolytic anemia. Warm antibody autoimmune hemolytic anemia. Idiopathic; Systemic lupus erythematosus (SLE) Evans syndrome (antiplatelet antibodies and hemolytic antibodies) Cold autoimmune hemolytic anemia Cold agglutinin disease; Paroxysmal cold hemoglobinuria (rare)