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SARS-CoV-2 is the virus that causes coronavirus disease 2019 (COVID-19). Some have been stated, to be of particular importance due to their potential for increased transmissibility, [1] increased virulence, or reduced effectiveness of vaccines against them. [2] [3] These variants contribute to the continuation of the COVID-19 pandemic.
Nonsense mutations comprise around 20% of single nucleotide substitutions within protein coding sequences that result in human disease. [12] Nonsense mutation-mediated pathology is often attributed to reduced amounts of full-length protein, because only 5-25% of transcripts possessing nonsense mutations do not undergo nonsense-mediated decay (NMD).
Here's what experts are saying about those findings and how they might affect the COVID-19 pandemic and efforts to develop vaccines and treatments.
Missense mutation is a type of nonsynonymous substitution in a DNA sequence. Two other types of nonsynonymous substitution are the nonsense mutations, in which a codon is changed to a premature stop codon that results in truncation of the resulting protein, and the nonstop mutations, in which a stop codon erasement results in a longer ...
The continued spread of the SARS-CoV-2 virus has spawned a Greek alphabet of variants - a naming system used by the World Health Organization to track concerning new mutations of the virus that ...
A mutation that speeds up COVID-19’s spread might explain why the virus—known as SARS-CoV-2—has so rapidly moved through North America and Europe, where the G614 mutated version is predominant.
The most commonly mutated protein within ORF1ab was papain-like protease (nsp3), and the single most commonly observed missense mutation was in RNA-dependent RNA polymerase. [13] Some PCR tests that detect COVID-19 analyze the specimen for the ORF1ab gene, among others. [14]
The government program that mailed free COVID-19 test kits to Americans came to an end when the CDC announced the end of the public health emergency in May 2023. But an iteration is coming back.
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