Search results
Results From The WOW.Com Content Network
The treatments to prevent the formation of blood clots are balanced against the risk of bleeding. [5] One of the goals of blood clot prevention is to limit venous stasis as this is a significant risk factor for forming blood clots in the deep veins of the legs. [6] Venous stasis can occur during the long periods of not moving.
Post-thrombotic syndrome (PTS), also called postphlebitic syndrome and venous stress disorder is a medical condition that may occur as a long-term complication of deep vein thrombosis (DVT). Signs and symptoms
Potential complications of venous stasis are: Venous ulcers; Blood clot formation in veins (venous thrombosis), that can occur in the deep veins of the legs (deep vein thrombosis, DVT) or in the superficial veins; Id reactions [1]
Chronic venous insufficiency. Varicose veins. Lymphedema. Cancer. A recent surgery, especially in the legs. A sedentary lifestyle. DVT can occur at any age, but being over the age of 60 increases ...
In orthopedic surgery, venous stasis can be temporarily provoked by a cessation of blood flow as part of the procedure. [56] Inactivity and immobilization contribute to venous stasis, as with orthopedic casts, [65] paralysis, sitting, long-haul travel, bed rest, hospitalization, [62] catatonia, [66] and in survivors of acute stroke. [67]
Compression therapy helps decrease venous pressure, prevents venous stasis and impairments of venous walls, and relieves heavy and aching legs. Knee-high compression stockings are used not only to help increase circulation, but also to help prevent the formation of blood clots in the lower legs.
Venous thrombosis can lead to pulmonary embolism when the migrated embolus becomes lodged in the lung. In people with a "shunt" (a connection between the pulmonary and systemic circulation), either in the heart or in the lung, a venous clot can also end up in the arteries and cause arterial embolism. [citation needed]
Venous thrombi are caused mainly by a combination of venous stasis and hypercoagulability—but to a lesser extent endothelial damage and activation. [7] The three factors of stasis, hypercoagulability, and alterations in the blood vessel wall represent Virchow's triad, and changes to the vessel wall are the least understood. [8]