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Alternative uses for statins for other health conditions are an ongoing area of research. Effects of statins on inflammation, cancer. Researchers of the current study note that chronic ...
IL-33 signals the body to produce other immune system cells when an injury or infection occurs. ... The mechanism through which statins affect cancer outcomes isn’t clear either. In Demehri’s ...
The trial focused on patients with normal low-density lipoprotein (LDL) cholesterol levels but increased levels of high-sensitivity C-reactive protein (hs-CRP). JUPITER was the first clinical trial to indicate that statin therapy may provide benefit to patients with low-to-normal LDL levels and no known cardiovascular disease.
Clinical studies have been conducted on the use of statins in dementia, [215] lung cancer, [216] nuclear cataracts, [217] hypertension, [218] [219] and prostate cancer [220] and breast cancer. [221] There is no high quality evidence that statins are useful for pneumonia . [ 222 ]
HMG-CoA reductase (3-hydroxy-3-methyl-glutaryl-coenzyme A reductase, official symbol HMGCR) is the rate-controlling enzyme (NADH-dependent, EC 1.1.1.88; NADPH-dependent, EC 1.1.1.34) of the mevalonate pathway, the metabolic pathway that produces cholesterol and other isoprenoids.
In 2020, Cancer Cell made a number of changes to increase diversity and inclusion of underrepresented minorities in cancer research and oncology. In June 2020, the journal announced the new members of its advisory board (previously editorial board), which includes a higher representation of female scientists (>40%) and physicians (~50%) and ...
Statins have sometimes been grouped into two groups of statins according to their structure. [9] Type 1 statins Statins that have substituted decalin-ring structure that resemble the first statin ever discovered, mevastatin have often been classified as type 1 statins due to their structural relationship. Statins that belong to this group are: [9]
The compound 8-Epi-xanthatin (EXT) is an extract from Xanthium fruit and is used as a natural transcription 3 inhibitor to induce apoptotic cell death and decrease DU-145 cancer cell proliferation. [8] DU-145 cells consistently express activated STAT3 and required STAT3 activity for proliferation and survival.